Deficiency in AMP-activated protein kinase exaggerates high fat diet-induced cardiac hypertrophy and contractile dysfunction - Wikidata - wikimedia - TriplyDB

Deficiency in AMP-activated protein kinase exaggerates high fat diet-induced cardiac hypertrophy and contractile dysfunction

Deficiency in AMP-activated protein kinase exaggerates high fat diet-induced cardiac hypertrophy and contractile dysfunction

http://www.wikidata.org/entity/Q34629075

about

Left ventricular mechanical dysfunction in diet-induced obese mice is exacerbated during inotropic stress: a cine DENSE cardiovascular magnetic resonance study Quantification of left ventricular volumes, mass, and ejection fraction using cine displacement encoding with stimulated echoes (DENSE) MRI Obesity reduces left ventricular strains, torsion, and synchrony in mouse models: a cine displacement encoding with stimulated echoes (DENSE) cardiovascular magnetic resonance study.Reproducibility of cine displacement encoding with stimulated echoes (DENSE) cardiovascular magnetic resonance for measuring left ventricular strains, torsion, and synchrony in mice.TRPV1 activation counters diet-induced obesity through sirtuin-1 activation and PRDM-16 deacetylation in brown adipose tissue.Mitochondrial aldehyde dehydrogenase (ALDH2) protects against streptozotocin-induced diabetic cardiomyopathy: role of GSK3β and mitochondrial function.Docosahexaenoic acid supplementation does not improve Western diet-induced cardiomyopathy in rats.Tauroursodeoxycholic acid mitigates high fat diet-induced cardiomyocyte contractile and intracellular Ca2+ anomalies.AMPK and insulin action--responses to ageing and high fat diet.The role of dietary fatty acids in predicting myocardial structure in fat-fed rats AMP-activated protein kinase deficiency rescues paraquat-induced cardiac contractile dysfunction through an autophagy-dependent mechanism Macrophage migration inhibitory factor (MIF) knockout preserves cardiac homeostasis through alleviating Akt-mediated myocardial autophagy suppression in high-fat diet-induced obesity.17-β estradiol attenuates ovariectomy-induced changes in cardiomyocyte contractile function via activation of AMP-activated protein kinase.Deficiency in AMPK attenuates ethanol-induced cardiac contractile dysfunction through inhibition of autophagosome formation.AMPK deficiency in cardiac muscle results in dilated cardiomyopathy in the absence of changes in energy metabolism.Punicalagin, an active component in pomegranate, ameliorates cardiac mitochondrial impairment in obese rats via AMPK activation Mechanisms of lipotoxicity in the cardiovascular system.Cardiac-specific knockout of ET(A) receptor mitigates low ambient temperature-induced cardiac hypertrophy and contractile dysfunction Partial Liver Kinase B1 (LKB1) Deficiency Promotes Diastolic Dysfunction, De Novo Systolic Dysfunction, Apoptosis, and Mitochondrial Dysfunction With Dietary Metabolic Challenge.Decreased levels of proapoptotic factors and increased key regulators of mitochondrial biogenesis constitute new potential beneficial features of long-lived growth hormone receptor gene-disrupted mice.Effects of short-chain acyl-CoA dehydrogenase on cardiomyocyte apoptosis.CYP2J2 and its metabolites (epoxyeicosatrienoic acids) attenuate cardiac hypertrophy by activating AMPKα2 and enhancing nuclear translocation of Akt1.Mammalian target of rapamycin (mTOR) inhibition with rapamycin improves cardiac function in type 2 diabetic mice: potential role of attenuated oxidative stress and altered contractile protein expression.AMP-activated protein kinase regulation and biological actions in the heart.mTOR signalling: the molecular interface connecting metabolic stress, aging and cardiovascular diseases.Influence of maternal overnutrition and gestational diabetes on the programming of metabolic health outcomes in the offspring: experimental evidence.Akt2 knockout mitigates chronic iNOS inhibition-induced cardiomyocyte atrophy and contractile dysfunction despite persistent insulin resistance.Insights for Oxidative Stress and mTOR Signaling in Myocardial Ischemia/Reperfusion Injury under Diabetes.Capsaicin induces browning of white adipose tissue and counters obesity by activating TRPV1 channel-dependent mechanisms.Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome.Inhibition of CYP2E1 attenuates chronic alcohol intake-induced myocardial contractile dysfunction and apoptosis.Inhibition of mammalian target of rapamycin protects against reperfusion injury in diabetic heart through STAT3 signaling.Maternal undernutrition around the time of conception and embryo number each impact on the abundance of key regulators of cardiac growth and metabolism in the fetal sheep heart.Differential effects of Mas receptor deficiency on cardiac function and blood pressure in obese male and female mice.MicroRNA Expression Signature Is Altered in the Cardiac Remodeling Induced by High Fat Diets.

P2860

Q30278803-D6316CF7-4611-4330-AC24-BDCB57E5B273 Q30371663-814B1B58-752C-49F4-ADB1-B55A5EE687A9 Q30415371-B2C82C12-7D41-44B2-BD22-389D0896F211 Q30417197-6BEA20E6-6E85-41DA-9003-63B12A6E1831 Q33627453-199CC295-44FC-4D0B-89F8-9A7A7F735D68 Q34242002-CF0E2AF6-08C6-4EEB-BD71-06C79D053980 Q34540353-F193EA2B-9147-49CD-9B4C-703D53DAD78F Q34718307-40F2D19A-401D-4803-BF4F-C3CB740D0C55 Q34720569-2038864F-2B72-49CE-B83B-571885AEB5FB Q35081224-D10FB83B-E550-4170-B424-AA641EE02AA3 Q35102608-A2B977C9-918E-40C6-A503-DFA6C0F4F5C6 Q35163560-A20192F0-0B51-4681-A527-1912F1C0F296 Q35602858-DE725A73-4FD3-4A15-81A6-D8E1979AC7A2 Q35965054-DD87998D-D52C-431A-B598-24A6C90FD69B Q36048973-EA193DF3-BB13-49A6-B139-028A154B5AB9 Q36274048-B2DB5DB9-17E2-433F-AB65-88245B2F94BF Q36377252-3E36A32B-CA3C-4B39-90B3-E26398705E50 Q36727926-DB8610BF-29FF-452D-B23E-D5FE11F02868 Q36877540-60C73305-5A9C-4FB6-8650-84177D52B849 Q37003167-0C78188F-920A-4A39-B29C-9D1F234F1EF9 Q37056975-185BF266-4581-4737-9603-19949D7D9376 Q37235552-CDEA4305-5656-4EBC-91DE-5AB12611BC8E Q37583506-C97F3636-7CF3-4386-8C5B-3157F30105E1 Q38039283-B7452FA6-607C-4AB7-A36F-F0E5A936273D Q38056232-A523C37A-6128-4A1A-A6AD-28F48917BA6B Q38352166-6A053919-CBC6-48ED-9021-1CE14F213993 Q38944416-2C783F6E-49E0-4800-B93B-D342222E5953 Q39180915-D3EDA9D0-6D7E-4BE6-B72E-B057B9272D5A Q41809091-246C23DC-76FC-483F-A255-F6DCF23AD3DB Q41820920-4AF0267E-A135-446D-8B35-E3A04CB2E5C8 Q42404252-92E29DF9-D01F-4D44-83C5-4FF592E0059A Q45012762-A96BDAC4-803F-4A09-B2D6-27D5B3AD48E7 Q46793766-95C1DC18-FAFA-461B-A081-D80B09E80F8F Q47915672-F79B95D1-2A6B-4DCF-BF37-418DDF97F2C8 Q51293120-07BF039E-E373-474E-A4C4-BA64A75B2D87

P2860

Deficiency in AMP-activated protein kinase exaggerates high fat diet-induced cardiac hypertrophy and contractile dysfunction

http://www.wikidata.org/entity/Q34629075

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@ast

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@en

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@nl

type

label

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@ast

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@en

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@nl

prefLabel

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@ast

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@en

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@nl

P1433

Q34629075-34C04DC8-9EBF-43FE-9C86-4385067B8BEA

P1476

Q34629075-21B9857B-22B5-4ACD-926D-B08F89AF5CBC

P2093

Q34629075-0AC2F4DF-5F7B-465E-9D93-98AB91757F59

Q34629075-11A9500B-C45B-437C-8957-AB9055F53A05

Q34629075-713C3FAC-3641-4AB3-B1ED-B57AED9FFAFD

Q34629075-72BE6AD5-2F37-4CC6-B63D-42DAB5145FEC

Q34629075-DBC4AEEF-C5E4-437B-A207-A59BCBB13501

Q34629075-E163C9F0-CEB9-42EC-A494-5EDDD779C2E6

P2860

Q34629075-0C0B0296-2B70-4A00-ACA8-311E39AFF5F4

Q34629075-14D51F57-730E-4E89-A99A-54F96F8B824C

Q34629075-1ADEF83C-F50F-499A-9024-7CCD4A61537D

Q34629075-1D5181FD-0F04-48E3-818B-070AD5D40AB2

Q34629075-1E266D43-7C26-4751-AB72-944AF92F630F

Q34629075-3DB1ECA0-C8C0-4781-885A-811C5B0A2F80

Q34629075-3EF24554-2AD7-4006-8D12-2EFC856E0245

Q34629075-42DB0D46-C413-4913-B809-616352FA02F3

Q34629075-48AD6609-F91C-4CA7-8386-522112563C0F

Q34629075-48B4A7E2-DA97-4FDE-AF44-4067A1FEEC8B

P304

Q34629075-0161D655-C8D9-4FAB-AEB5-06DDE4CE4633

P31

Q34629075-523AFF1B-FDEF-4253-BA9A-94D2C013B591

P356

Q34629075-EDD39F4E-32AA-41A6-9DA2-AD842D4D600A

P433

Q34629075-BEF59832-259C-4C9F-A664-19B02BB2A38B

P478

Q34629075-3FADBD27-1AF2-4350-935C-815FC63D75B1

P577

Q34629075-D579C0C3-FF3B-4053-9561-DECF6805CAE5

P5875

Q34629075-6252A808-6E30-4867-AC66-180AB562879A

P698

Q34629075-E92750C3-F48F-4E47-83EC-EB5C16CF6088

P932

Q34629075-659A5CFC-F250-4B28-A00E-2F23CCF008DF

P356

J.YJMCC.2010.12.007

P1433

Journal of Molecular and Cellular Cardiology

P1476

Deficiency in AMP-activated pr ...... hy and contractile dysfunction

@en

P2093

Anna F Huff

http://www.w3.org/2001/XMLSchema#string

Jun Ren

http://www.w3.org/2001/XMLSchema#string

Junxing Zhao

http://www.w3.org/2001/XMLSchema#string

Machender R Kandadi

http://www.w3.org/2001/XMLSchema#string

Min Du

http://www.w3.org/2001/XMLSchema#string

Subat Turdi

http://www.w3.org/2001/XMLSchema#string

P2860

The AMP-activated protein kinase alpha2 catalytic subunit controls whole-body insulin sensitivity

Cardiomyocyte cyclooxygenase-2 influences cardiac rhythm and function

A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle

Inhibition of GLUT4 translocation by Tbc1d1, a Rab GTPase-activating protein abundant in skeletal muscle, is partially relieved by AMP-activated protein kinase activation.

AICA riboside increases AMP-activated protein kinase, fatty acid oxidation, and glucose uptake in rat muscle.

Emerging role for AS160/TBC1D4 and TBC1D1 in the regulation of GLUT4 traffic

Chromium (D-phenylalanine)3 supplementation alters glucose disposal, insulin signaling, and glucose transporter-4 membrane translocation in insulin-resistant mice.

Obesity as a cardiovascular risk factor.

The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease.

AMP-activated protein kinase: the guardian of cardiac energy status.

P304

712-722

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.YJMCC.2010.12.007

http://www.w3.org/2001/XMLSchema#string

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

50

http://www.w3.org/2001/XMLSchema#string

P577

2010-12-16T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

49692576

http://www.w3.org/2001/XMLSchema#string

P698

21167835

http://www.w3.org/2001/XMLSchema#string

P932

3049828

http://www.w3.org/2001/XMLSchema#string