Akt2 knockout mitigates chronic iNOS inhibition-induced cardiomyocyte atrophy and contractile dysfunction despite persistent insulin resistance.
about
Akt2 knockout alleviates prolonged caloric restriction-induced change in cardiac contractile function through regulation of autophagy.Ablation of Akt2 protects against lipopolysaccharide-induced cardiac dysfunction: role of Akt ubiquitination E3 ligase TRAF6.Targeted deletion of PTEN in cardiomyocytes renders cardiac contractile dysfunction through interruption of Pink1-AMPK signaling and autophagyAutophagy in sepsis: Degradation into exhaustion?Contractile Activity Regulates Inducible Nitric Oxide Synthase Expression and NO(i) Production in Cardiomyocytes via a FAK-Dependent Signaling PathwayFolic acid reverses nitric oxide synthase uncoupling and prevents cardiac dysfunction in insulin resistance: role of Ca2+/calmodulin-activated protein kinase II.
P2860
Akt2 knockout mitigates chronic iNOS inhibition-induced cardiomyocyte atrophy and contractile dysfunction despite persistent insulin resistance.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Akt2 knockout mitigates chroni ...... persistent insulin resistance.
@en
type
label
Akt2 knockout mitigates chroni ...... persistent insulin resistance.
@en
prefLabel
Akt2 knockout mitigates chroni ...... persistent insulin resistance.
@en
P2860
P1433
P1476
Akt2 knockout mitigates chroni ...... persistent insulin resistance.
@en
P2093
P2860
P304
P356
10.1016/J.TOXLET.2011.09.015
P577
2011-09-22T00:00:00Z