The interleukin-17 receptor B subunit is essential for the Th2 response to Helicobacter pylori, but not for control of bacterial burden.
about
Metabolic consequences of Helicobacter pylori infection and eradication.Systems modeling of the role of interleukin-21 in the maintenance of effector CD4+ T cell responses during chronic Helicobacter pylori infection.Immunity, inflammation, and vaccines for Helicobacter pylori.Immunology and vaccines and nanovaccines for Helicobacter pylori infection.Early Molecular Events in Murine Gastric Epithelial Cells Mediated by Helicobacter pylori CagA.
P2860
The interleukin-17 receptor B subunit is essential for the Th2 response to Helicobacter pylori, but not for control of bacterial burden.
description
2013 nî lūn-bûn
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2013 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մարտին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
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2013年论文
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name
The interleukin-17 receptor B ...... r control of bacterial burden.
@ast
The interleukin-17 receptor B ...... r control of bacterial burden.
@en
The interleukin-17 receptor B ...... r control of bacterial burden.
@nl
type
label
The interleukin-17 receptor B ...... r control of bacterial burden.
@ast
The interleukin-17 receptor B ...... r control of bacterial burden.
@en
The interleukin-17 receptor B ...... r control of bacterial burden.
@nl
prefLabel
The interleukin-17 receptor B ...... r control of bacterial burden.
@ast
The interleukin-17 receptor B ...... r control of bacterial burden.
@en
The interleukin-17 receptor B ...... r control of bacterial burden.
@nl
P2093
P2860
P1433
P1476
The interleukin-17 receptor B ...... r control of bacterial burden.
@en
P2093
Dennis J Horvath
Jana N Radin
M Kay Washington
Sung Hoon Cho
P2860
P304
P356
10.1371/JOURNAL.PONE.0060363
P407
P577
2013-03-22T00:00:00Z