Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency. - Wikidata - wikimedia - TriplyDB

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

http://www.wikidata.org/entity/Q34648219

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Immunology in the clinic review series; focus on type 1 diabetes and viruses: role of antibodies enhancing the infection with Coxsackievirus-B in the pathogenesis of type 1 diabetes Infection as a cause of type 1 diabetes?The microbiology of human hygiene and its impact on type 1 diabetes Potential viral pathogenic mechanism in human type 1 diabetes Molecular basis for a lack of correlation between viral fitness and cell killing capacity Do viral infections protect from or enhance type 1 diabetes and how can we tell the difference?Beta-cell specific production of IL6 in conjunction with a mainly intracellular but not mainly surface viral protein causes diabetes.Type I diabetes mellitus: genetic factors and presumptive enteroviral etiology or protection.Toll-like receptor 3 is critical for coxsackievirus B4-induced type 1 diabetes in female NOD mice.Recent progress in understanding coxsackievirus replication, dissemination, and pathogenesis.Cytokine and chemokine production by human pancreatic islets upon enterovirus infection Coxsackievirus infections and NOD mice: relevant models of protection from, and induction of, type 1 diabetes.Induction of an antiviral state and attenuated coxsackievirus replication in type III interferon-treated primary human pancreatic islets.Protection against type 1 diabetes upon Coxsackievirus B4 infection and iNKT-cell stimulation: role of suppressive macrophages.Protection against or triggering of Type 1 diabetes? Different roles for viral infections Viruses and cytotoxic T lymphocytes in type 1 diabetes.Prevention or acceleration of type 1 diabetes by viruses.Enteroviruses, hygiene and type 1 diabetes: toward a preventive vaccine.Differential effects of three echovirus strains on cell lysis and insulin secretion in beta cell derived lines.Coxsackievirus can persist in murine pancreas by deletion of 5' terminal genomic sequences.Enteroviruses and type 1 diabetes.99th Dahlem conference on infection, inflammation and chronic inflammatory disorders: viruses, autoimmunity and immunoregulation.In silico and ex vivo approaches indicate immune pressure on capsid and non-capsid regions of coxsackie B viruses in the human system.

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P2860

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

http://www.wikidata.org/entity/Q34648219

description

2006 nî lūn-bûn

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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2006 թվականի հունիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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type

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

@ast

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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P1433

Journal of Virology

P1476

Group B coxsackievirus diabetogenic phenotype correlates with replication efficiency.

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P2093

Ken Kono

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Kisoon Kim

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Kristen M Drescher

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Steven Tracy

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Toru Kanno

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P2860

Opinion: Satisfaction (not) guaranteed: re-evaluating the use of animal models of type 1 diabetes

Persistent infection of human pancreatic islets by coxsackievirus B is associated with alpha interferon synthesis in beta cells.

Environmental factors in the pathogenesis of type 1 diabetes mellitus.

Genomic determinants of cardiovirulence in coxsackievirus B3 clinical isolates: localization to the 5' nontranslated region.

The NOD mouse model of type 1 diabetes: as good as it gets?

Echovirus 4 and type 1 diabetes mellitus.

Toward testing the hypothesis that group B coxsackieviruses (CVB) trigger insulin-dependent diabetes: inoculating nonobese diabetic mice with CVB markedly lowers diabetes incidence.

Environmental factors in the etiology of type 1 diabetes.

The role of viruses in the pathogenesis of pancreatic disease and diabetes mellitus.

RNA recombination plays a major role in genomic change during circulation of coxsackie B viruses.

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5637-5643

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10.1128/JVI.02361-05

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11

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80

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