Lack of PPARα exacerbates lipopolysaccharide-induced liver toxicity through STAT1 inflammatory signaling and increased oxidative/nitrosative stress

Lack of PPARα exacerbates lipopolysaccharide-induced liver toxicity through STAT1 inflammatory signaling and increased oxidative/nitrosative stress

Item
http://www.wikidata.org/entity/Q34664393
Mitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modificationsFunctional roles of protein nitration in acute and chronic liver diseasesPost-translational modifications of mitochondrial aldehyde dehydrogenase and biomedical implications.Peroxisome proliferator-activated receptor α activation attenuates the inflammatory response to protect the liver from acute failure by promoting the autophagy pathway.The Mechanism of Interferon Refractoriness During Hepatitis C Virus Infection and Its Reversal with a Peroxisome Proliferator-Activated Receptor α AgonistOmega-9 Oleic Acid Induces Fatty Acid Oxidation and Decreases Organ Dysfunction and Mortality in Experimental Sepsis.Inhibition of glycogen synthase kinase 3β promotes autophagy to protect mice from acute liver failure mediated by peroxisome proliferator-activated receptor α.Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injuryActivation of PPARα by Wy-14643 ameliorates systemic lipopolysaccharide-induced acute lung injury.Acyl-CoA Thioesterase 1 (ACOT1) Regulates PPARα to Couple Fatty Acid Flux With Oxidative Capacity During Fasting.Peroxisome proliferator-activated receptor alpha mediates C/EBP homologous protein to protect mice from acute liver failure.Hepatocyte-specific PPARA expression exclusively promotes agonist-induced cell proliferation without influence from nonparenchymal cells.Furosine Induced Apoptosis by the Regulation of STAT1/STAT2 and UBA7/UBE2L6 Genes in HepG2 Cells.Role of adipose triglyceride lipase (PNPLA2) in protection from hepatic inflammation in mouse models of steatohepatitis and endotoxemia
P2860

Lack of PPARα exacerbates lipopolysaccharide-induced liver toxicity through STAT1 inflammatory signaling and increased oxidative/nitrosative stress

Item
http://www.wikidata.org/entity/Q34664393
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@ast
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@en
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@nl
type
label
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@ast
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@en
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@nl
prefLabel
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@ast
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@en
Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@nl
P1433
P1476
P2093
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P356
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Lack of PPARα exacerbates lipo ...... d oxidative/nitrosative stress
@en
P2093
P2860
P304
P31
P356
10.1016/J.TOXLET.2011.01.013
P433
P478
P577
P5875
P698
P921
P932