mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers. - Wikidata - wikimedia - TriplyDB

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

http://www.wikidata.org/entity/Q34673900

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Temsirolimus inhibits proliferation and migration in retinal pigment epithelial and endothelial cells via mTOR inhibition and decreases VEGF and PDGF expression Pilot study of sirolimus in patients with PIK3CA mutant/amplified refractory solid cancer.Clinical sequencing using a next-generation sequencing-based multiplex gene assay in patients with advanced solid tumors.Selective anti-cancer agents as anti-aging drugs A phase 1 study of 131I-CLR1404 in patients with relapsed or refractory advanced solid tumors: dosimetry, biodistribution, pharmacokinetics, and safety.Ribosomal protein S27-like in colorectal cancer: a candidate for predicting prognoses.Colon Cancer Tumorigenesis Initiated by the H1047R Mutant PI3K.Metformin use and the risk of colorectal adenoma: A systematic review and meta-analysis.Targeting mTOR network in colorectal cancer therapy Rodent intestinal epithelial carcinogenesis: pathology and preclinical models.Effects of RAL signal transduction in KRAS- and BRAF-mutated cells and prognostic potential of the RAL signature in colorectal cancer Suppression of the proliferation of hypoxia-Induced retinal pigment epithelial cell by rapamycin through the /mTOR/HIF-1α/VEGF/ signaling.

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mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

http://www.wikidata.org/entity/Q34673900

description

2013 nî lūn-bûn

@nan

2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

@wuu

name

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@ast

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@en

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@nl

type

label

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@ast

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@en

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@nl

prefLabel

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@ast

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@en

mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

@nl

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mTOR inhibition elicits a dramatic response in PI3K-dependent colon cancers.

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Alyssa A Leystra

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Dawn Albrecht

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Dustin A Deming

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Jamey P Weichert

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Laura Nettekoven

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Linda Clipson

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Mary Kay Washington

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Mohammed Farhoud

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Richard B Halberg

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Ruth Sullivan

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P2860

S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

Molecular origins of cancer: Molecular basis of colorectal cancer

Targeting the PI3K signaling pathway in cancer

Effective use of PI3K and MEK inhibitors to treat mutant Kras G12D and PIK3CA H1047R murine lung cancers.

Anaplastic lymphoma kinase inhibition in non-small-cell lung cancer.

Inhibition of Mutated, Activated BRAF in Metastatic Melanoma

High frequency of mutations of the PIK3CA gene in human cancers

Rapamycin-FKBP specifically blocks growth-dependent activation of and signaling by the 70 kd S6 protein kinases

Genetic alterations during colorectal-tumor development

The phosphatidylinositol 3-Kinase AKT pathway in human cancer

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e60709

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10.1371/JOURNAL.PONE.0060709

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