Muscarinic receptor signaling in the pathophysiology of asthma and COPD. - Wikidata - wikimedia - TriplyDB

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

http://www.wikidata.org/entity/Q34684230

about

Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus higher dose ICS for adults with asthma Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus addition of long-acting beta2-agonists (LABA) for adults with asthma Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus addition of long-acting beta 2 -agonists (LABA) for adults with asthma Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus higher dose ICS for adults with asthma Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus the same dose of ICS alone for adults with asthma Long-acting muscarinic antagonists (LAMA) added to inhaled corticosteroids (ICS) versus the same dose of ICS for adults with asthma Regulator of G Protein Signaling 2: A Versatile Regulator of Vascular Function Ten years of tiotropium: clinical impact and patient perspectives Long-acting muscarinic receptor antagonists for the treatment of chronic airway diseases Mapping physiological G protein-coupled receptor signaling pathways reveals a role for receptor phosphorylation in airway contraction The cellular and molecular basis of bitter tastant-induced bronchodilation Tiotropium in asthma: what is the evidence and how does it fit in?Caveolin-1 knockout mice exhibit airway hyperreactivity Kaempferol Inhibits Endoplasmic Reticulum Stress-Associated Mucus Hypersecretion in Airway Epithelial Cells And Ovalbumin-Sensitized Mice Muscarinic antagonists in early stage clinical development for the treatment of asthma.Muscarinic receptor-mediated bronchoconstriction is coupled to caveolae in murine airways The evidence on tiotropium bromide in asthma: from the rationale to the bedside.Crosstalk between beta-2-adrenoceptor and muscarinic acetylcholine receptors in the airway Pro-inflammatory mechanisms of muscarinic receptor stimulation in airway smooth muscle Differential expression analysis for pathways.Detrimental effects of albuterol on airway responsiveness requires airway inflammation and is independent of β-receptor affinity in murine models of asthma Expression of functional leukotriene B4 receptors on human airway smooth muscle cells.Muscarinic M3 receptors on structural cells regulate cigarette smoke-induced neutrophilic airway inflammation in mice Tiotropium in asthma: a systematic review Long-term exposure to muscarinic agonists decreases expression of contractile proteins and responsiveness of rabbit tracheal smooth muscle cells mAChRs activation induces epithelial-mesenchymal transition on lung epithelial cells Epithelium integrity is crucial for the relaxant activity of brain natriuretic peptide in human isolated bronchi.Suppression of IL-8 production from airway cells by tiotropium bromide in vitro.Maternal exposure to secondhand cigarette smoke primes the lung for induction of phosphodiesterase-4D5 isozyme and exacerbated Th2 responses: rolipram attenuates the airway hyperreactivity and muscarinic receptor expression but not lung inflammation Targeted lung denervation for moderate to severe COPD: a pilot study.β2-Adrenoceptor agonist-induced RGS2 expression is a genomic mechanism of bronchoprotection that is enhanced by glucocorticoids Overcoming beta-agonist tolerance: high dose salbutamol and ipratropium bromide. Two randomised controlled trials Circulating Human Eosinophils Share a Similar Transcriptional Profile in Asthma and Other Hypereosinophilic Disorders.The role of tiotropium in the management of asthma.Impact of Long-Term Tiotropium Bromide Therapy on Annual Lung Function Decline in Adult Patients with Cystic Fibrosis Regional quantification of muscarinic acetylcholine receptors and β-adrenoceptors in human airways.Luminal cholinergic signalling in airway lining fluid: a novel mechanism for activating chloride secretion via Ca²⁺-dependent Cl⁻ and K⁺ channels.Circadian disruption alters mouse lung clock gene expression and lung mechanics Protective Roles for RGS2 in a Mouse Model of House Dust Mite-Induced Airway Inflammation.Impact of GPCRs in clinical medicine: monogenic diseases, genetic variants and drug targets

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

http://www.wikidata.org/entity/Q34684230

description

2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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type

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

@ast

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

@ast

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

@en

Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Muscarinic receptor signaling in the pathophysiology of asthma and COPD.

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Herman Meurs

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Johan Zaagsma

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Reinoud Gosens

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P2860

Involvement of RhoA-mediated Ca2+ sensitization in antigen-induced bronchial smooth muscle hyperresponsiveness in mice

Pharmacology of airway afferent nerve activity

Upregulation of mRNA encoding the M5 muscarinic acetylcholine receptor in human T- and B-lymphocytes during immunological responses

Augmented acetylcholine-induced translocation of RhoA in bronchial smooth muscle from antigen-induced airway hyperresponsive rats

Ca2+ sensitivity of smooth muscle and nonmuscle myosin II: modulated by G proteins, kinases, and myosin phosphatase

The nature of small-airway obstruction in chronic obstructive pulmonary disease

Changes in biophysical and biochemical properties of single bronchial smooth muscle cells from asthmatic subjects.

Expression and function of the non-neuronal cholinergic system in endothelial cells.

Hyperplasia of smooth muscle in mild to moderate asthma without changes in cell size or gene expression.

Ca2+ oscillation and c-fos gene expression induced via muscarinic acetylcholine receptor in human T- and B-cell lines.

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10.1186/1465-9921-7-73

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Andrew J Halayko

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pathophysiology

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membrane proteins

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