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Microbes bind complement inhibitor factor H via a common site

Microbes bind complement inhibitor factor H via a common site

http://www.wikidata.org/entity/Q34699715

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Molecular basis of host specificity in human pathogenic bacteria Structural Basis for Complement Evasion by Lyme Disease Pathogen Borrelia burgdorferi Multifunctional and Redundant Roles of Borrelia burgdorferi Outer Surface Proteins in Tissue Adhesion, Colonization, and Complement Evasion Complement System Part II: Role in Immunity The major autoantibody epitope on factor H in atypical hemolytic uremic syndrome is structurally different from its homologous site in factor H-related protein 1, supporting a novel model for induction of autoimmunity in this disease.Self-nonself discrimination by the complement system.Anti-complement-factor H-associated glomerulopathies.Immune evasion of Borrelia miyamotoi: CbiA, a novel outer surface protein exhibiting complement binding and inactivating properties.Factor H Family Proteins in Complement Evasion of Microorganisms.The Borrelia hermsii factor H binding protein FhbA is not required for infectivity in mice or for resistance to human complement in vitro.Delineating the importance of serum opsonins and the bacterial capsule in affecting the uptake and killing of Burkholderia pseudomallei by murine neutrophils and macrophages Interactions of Candida albicans with host epithelial surfaces Anopheles midgut epithelium evades human complement activity by capturing factor H from the blood meal.Complement evasion by Bordetella pertussis: implications for improving current vaccines Mechanisms of Bone Resorption in Periodontitis.FHR3 Blocks C3d-Mediated Coactivation of Human B Cells.Recruitment of Factor H to the Streptococcus suis Cell Surface is Multifactorial Evasion and interactions of the humoral innate immune response in pathogen invasion, autoimmune disease, and cancer Staphylococcal protein Ecb impairs complement receptor-1 mediated recognition of opsonized bacteria.Complement Factor H Binds to Human Serum Apolipoprotein E and Mediates Complement Regulation on High Density Lipoprotein Particles.A Novel Factor H-Fc Chimeric Immunotherapeutic Molecule against Neisseria gonorrhoeae.Defining the Binding Region in Factor H to Develop a Therapeutic Factor H-Fc Fusion Protein against Non-Typeable Haemophilus influenzae Hijacking Complement Regulatory Proteins for Bacterial Immune Evasion.That's my story, and I'm sticking to it--an update on B. burgdorferi adhesins.Low-dose recombinant properdin provides substantial protection against Streptococcus pneumoniae and Neisseria meningitidis infection.Importance of (antibody-dependent) complement-mediated serum killing in protection against Bordetella pertussis.Streptococcus pneumoniae proteomics: determinants of pathogenesis and vaccine development.Utilizing complement evasion strategies to design complement-based antibacterial immunotherapeutics: Lessons from the pathogenic Neisseriae.Pentraxins in the activation and regulation of innate immunity.Complement factor H in host defense and immune evasion.Complement Evasion by Pathogenic Leptospira.There Is a Method to the Madness: Strategies to Study Host Complement Evasion by Lyme Disease and Relapsing Fever Spirochetes The outer membrane protease PgtE of Salmonella enterica interferes with the alternative complement pathway by cleaving factors B and H.The mammalian complement system as an epitome of host-pathogen genetic conflicts.Conserved Patterns of Microbial Immune Escape: Pathogenic Microbes of Diverse Origin Target the Human Terminal Complement Inhibitor Vitronectin via a Single Common Motif.The Bordetella pertussis Bps polysaccharide enhances lung colonization by conferring protection from complement-mediated killing.The Plasmodium falciparum blood stages acquire factor H family proteins to evade destruction by human complement.Binding of human factor H to outer membrane protein P5 of non-typeable Haemophilus influenzae contributes to complement resistance Neisseria cinerea expresses a functional factor H binding protein which is recognised by immune responses elicited by meningococcal vaccines.Outer membrane protein OlpA contributes to Moraxella catarrhalis serum resistance via interaction with factor H and the alternative pathway.

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Microbes bind complement inhibitor factor H via a common site

http://www.wikidata.org/entity/Q34699715

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2013年の論文

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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Microbes bind complement inhibitor factor H via a common site

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A Goldman

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H Amdahl

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P2860

Structure of complement factor H carboxyl-terminus reveals molecular basis of atypical haemolytic uremic syndrome.

Control of the amplification convertase of complement by the plasma protein beta1H

Structural basis for engagement by complement factor H of C3b on a self surface

Dual interaction of factor H with C3d and glycosaminoglycans in host-nonhost discrimination by complement

Structure of complement fragment C3b-factor H and implications for host protection by complement regulators

Structure shows that a glycosaminoglycan and protein recognition site in factor H is perturbed by age-related macular degeneration-linked single nucleotide polymorphism

Neisseria meningitidis recruits factor H using protein mimicry of host carbohydrates

Both domain 19 and domain 20 of factor H are involved in binding to complement C3b and C3d

Complement factor H variant increases the risk of age-related macular degeneration

Streptococcal protein G. Gene structure and protein binding properties

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