Type III secretion-dependent modulation of innate immunity as one of multiple factors regulated by Pseudomonas aeruginosa RetS.
about
A type VI secretion system of Pseudomonas aeruginosa targets a toxin to bacteriaIn vivo imaging and genetic analysis link bacterial motility and symbiosis in the zebrafish gut.Pseudomonas aeruginosa induces membrane blebs in epithelial cells, which are utilized as a niche for intracellular replication and motilityPseudomonas aeruginosa Type III secretion system interacts with phagocytes to modulate systemic infection of zebrafish embryosCytotoxic clinical isolates of Pseudomonas aeruginosa identified during the Steroids for Corneal Ulcers Trial show elevated resistance to fluoroquinolones.The impact of inoculation parameters on the pathogenesis of contact lens-related infectious keratitis.Pseudomonas aeruginosa utilizes the type III secreted toxin ExoS to avoid acidified compartments within epithelial cellsThe importance of the Pseudomonas aeruginosa type III secretion system in epithelium traversal depends upon conditions of host susceptibilityPathogenesis of contact lens-associated microbial keratitisAdenylate cyclase activity of Pseudomonas aeruginosa ExoY can mediate bleb-niche formation in epithelial cells and contributes to virulence.SagS contributes to the motile-sessile switch and acts in concert with BfiSR to enable Pseudomonas aeruginosa biofilm formationStructural Analysis of the Regulatory Domain of ExsA, a Key Transcriptional Regulator of the Type Three Secretion System in Pseudomonas aeruginosaTraversal of multilayered corneal epithelia by cytotoxic Pseudomonas aeruginosa requires the phospholipase domain of exoU.Pseudomonas aeruginosa RsmA plays an important role during murine infection by influencing colonization, virulence, persistence, and pulmonary inflammationTranslocon-independent intracellular replication by Pseudomonas aeruginosa requires the ADP-ribosylation domain of ExoS.Self-trimerization of ExsD limits inhibition of the Pseudomonas aeruginosa transcriptional activator ExsA in vitroWhy does the healthy cornea resist Pseudomonas aeruginosa infection?Association between cytotoxic and invasive Pseudomonas aeruginosa and clinical outcomes in bacterial keratitisSmall RNAs as regulators of primary and secondary metabolism in Pseudomonas species.Mutation of the phospholipase catalytic domain of the Pseudomonas aeruginosa cytotoxin ExoU abolishes colonization promoting activity and reduces corneal disease severity.Clearance of Pseudomonas aeruginosa from a healthy ocular surface involves surfactant protein D and is compromised by bacterial elastase in a murine null-infection model.
P2860
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P2860
Type III secretion-dependent modulation of innate immunity as one of multiple factors regulated by Pseudomonas aeruginosa RetS.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@ast
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@en
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@nl
type
label
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@ast
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@en
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@nl
prefLabel
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@ast
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@en
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@nl
P2093
P2860
P356
P1476
Type III secretion-dependent m ...... y Pseudomonas aeruginosa RetS.
@en
P2093
David J Evans
Irandokht Zolfaghar
Reza Ronaghi
Suzanne M J Fleiszig
P2860
P304
P356
10.1128/IAI.01891-05
P407
P577
2006-07-01T00:00:00Z