CSNK1α1 mediates malignant plasma cell survival. - Wikidata - wikimedia - TriplyDB

CSNK1α1 mediates malignant plasma cell survival.

CSNK1α1 mediates malignant plasma cell survival.

http://www.wikidata.org/entity/Q34774056

about

Casein kinase 1α-dependent feedback loop controls autophagy in RAS-driven cancers.Discovery and Optimization of Macrocyclic Quinoxaline-pyrrolo-dihydropiperidinones as Potent Pim-1/2 Kinase Inhibitors.Inactivation of CK1α in multiple myeloma empowers drug cytotoxicity by affecting AKT and β-catenin survival signaling pathways.Casein kinases as potential therapeutic targets.Lenalidomide increases human dendritic cell maturation in multiple myeloma patients targeting monocyte differentiation and modulating mesenchymal stromal cell inhibitory properties CK1δ in lymphoma: gene expression and mutation analyses and validation of CK1δ kinase activity for therapeutic application.Role of protein kinases CK1α and CK2 in multiple myeloma: regulation of pivotal survival and stress-managing pathways.Casein kinase 1α: biological mechanisms and theranostic potential.

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CSNK1α1 mediates malignant plasma cell survival.

http://www.wikidata.org/entity/Q34774056

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

@zh-tw

2014年论文

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name

CSNK1α1 mediates malignant plasma cell survival.

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CSNK1α1 mediates malignant plasma cell survival.

@en

CSNK1α1 mediates malignant plasma cell survival.

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type

label

CSNK1α1 mediates malignant plasma cell survival.

@ast

CSNK1α1 mediates malignant plasma cell survival.

@en

CSNK1α1 mediates malignant plasma cell survival.

@nl

prefLabel

CSNK1α1 mediates malignant plasma cell survival.

@ast

CSNK1α1 mediates malignant plasma cell survival.

@en

CSNK1α1 mediates malignant plasma cell survival.

@nl

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CSNK1α1 mediates malignant plasma cell survival.

@en

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D Cirstea

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D Lu

http://www.w3.org/2001/XMLSchema#string

K C Anderson

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N C Munshi

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W Song

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P2860

The casein kinase 1 family: participation in multiple cellular processes in eukaryotes

Posttranslational modification of MDM2

Frequent translocation t(4;14)(p16.3;q32.3) in multiple myeloma is associated with increased expression and activating mutations of fibroblast growth factor receptor 3

BET bromodomain inhibition as a therapeutic strategy to target c-Myc

A novel chromosomal translocation t(4; 14)(p16.3; q32) in multiple myeloma involves the fibroblast growth-factor receptor 3 gene

Phosphorylation of FADD at serine 194 by CKIalpha regulates its nonapoptotic activities

Intramolecular masking of nuclear import signal on NF-AT4 by casein kinase I and MEKK1

A novel small molecule inhibitor of deubiquitylating enzyme USP14 and UCHL5 induces apoptosis in multiple myeloma and overcomes bortezomib resistance

Exploiting the MDM2-CK1α protein-protein interface to develop novel biologics that induce UBL-kinase-modification and inhibit cell growth

Velcade: U.S. FDA approval for the treatment of multiple myeloma progressing on prior therapy.

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10.1038/LEU.2014.202

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24962017

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