Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathy. - Wikidata - wikimedia - TriplyDB

Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathy.

Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathy.

http://www.wikidata.org/entity/Q34789824

about

Glucose Transporters in Cardiac Metabolism and Hypertrophy Glucose Transporter Type I Deficiency (G1D) at 25 (1990-2015): Presumptions, Facts, and the Lives of Persons With This Rare Disease Glucose transporter isoform-3 mutations cause early pregnancy loss and fetal growth restriction Nephron-deficient Fvb mice develop rapidly progressive renal failure and heavy albuminuria involving excess glomerular GLUT1 and VEGF.Glut1 deficiency (G1D): epilepsy and metabolic dysfunction in a mouse model of the most common human phenotype.Neuronal glucose transporter isoform 3 deficient mice demonstrate features of autism spectrum disorders Minireview: Metabolism of female reproduction: regulatory mechanisms and clinical implications Expression of glucose transporter 1 confers susceptibility to human T-cell leukemia virus envelope-mediated fusion.Murine Glut-1 transporter haploinsufficiency: postnatal deceleration of brain weight and reactive astrocytosis.Diet-induced obesity model: abnormal oocytes and persistent growth abnormalities in the offspring.Prenatal caloric restriction enhances DNA methylation and MeCP2 recruitment with reduced murine placental glucose transporter isoform 3 expression Hypoxic-ischemic brain injury exacerbates neuronal apoptosis and precipitates spontaneous seizures in glucose transporter isoform 3 heterozygous null mice.Cell metabolism: an essential link between cell growth and apoptosis Epidemiologic and genetic aspects of spina bifida and other neural tube defects.Regulation of facilitative glucose transporters and AKT/MAPK/PRKAA signaling via estradiol and progesterone in the mouse uterine epithelium A 10 bp deletion polymorphism and 2 new variations in the GLUT1 gene associated with meningomyelocele.GLUT1 enhances mTOR activity independently of TSC2 and AMPK.Cytokine stimulation promotes glucose uptake via phosphatidylinositol-3 kinase/Akt regulation of Glut1 activity and trafficking.Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions.Polony analysis of gene expression in ES cells and blastocysts.Hyperglycemia slows embryonic growth and suppresses cell cycle via cyclin D1 and p21.Effects of glucose metabolism during in vitro maturation on cytoplasmic maturation of mouse oocytes Genetic mouse models to study blood-brain barrier development and function Genes in glucose metabolism and association with spina bifida In the search for genes responsible for neural tube defects, glucose metabolism matters.Family-based association study between SLC2A1, HK1, and LEPR polymorphisms with myelomeningocele in Chile.Will the original glucose transporter isoform please stand up!Glucose transporters in gametes and preimplantation embryos.Genetic basis of susceptibility to teratogen induced birth defects.Genetic studies of myelomeningocele.Glucose transporter 1-mediated glucose uptake is limiting for B-cell acute lymphoblastic leukemia anabolic metabolism and resistance to apoptosis.Molecular diagnosis of vertebral segmentation disorders in humans.Maternal Calorie Restriction Causing Uteroplacental Insufficiency Differentially Affects Mammalian Placental Glucose and Leucine Transport Molecular Mechanisms.A mouse model for Glut-1 haploinsufficiency.GLUT3 gene expression is critical for embryonic growth, brain development and survival.Glucose transporter-1 in the hypothalamic glial cells mediates glucose sensing to regulate glucose production in vivo Mutations in the SLC2A10 gene cause arterial abnormalities in mice.Phosphatidylinositol 3-kinase activity is critical for glucose metabolism and embryo survival in murine blastocysts.High glucose-induced inhibition of 2-deoxyglucose uptake is mediated by cAMP, protein kinase C, oxidative stress and mitogen-activated protein kinases in mouse embryonic stem cells.Effects of in vitro oocyte maturation and embryo culture on the expression of glucose transporters, glucose metabolism and insulin signaling genes in rhesus monkey oocytes and preimplantation embryos.

P2860

Q28075863-3EF50D00-798A-467D-A562-D228E2C1CC57 Q28082363-9BF935EB-6FCD-403D-A49F-277D735EC181 Q28594333-5735D482-A73F-4128-9CF5-EC206BA2D507 Q30273921-C3299464-C798-4525-986B-C9AB2ADB2618 Q30448770-382D5B52-7EDD-4164-BF23-CB2DC846593D Q30596233-110DBF77-1E3F-4CC1-90DB-E8B85B4177EB Q33701382-AD2BE8EE-AA82-44C1-967D-3EB25459A7A8 Q33707390-9C9B06EF-355A-48C6-B6B2-8F7F495B36C0 Q34090076-EC641BF6-0FE9-4E32-B6CB-7957E5785804 Q34132433-6FE4A3D6-7F6F-442C-97F8-32440C9CF13F Q34401375-8F27FA89-1379-4431-B794-2C00042C3D83 Q34401387-1B778125-645B-49B1-A43D-44E60C8974DE Q34440592-6FB6AF32-1344-4A22-928D-7C0623840F34 Q34645951-589C876B-E566-47DE-B532-BE7277DEE4C3 Q34979443-71575120-E241-4DED-BFDD-A7DD91A1ADE2 Q35051429-8073BD9D-7A42-4271-9AFD-A43662112B5F Q35216825-F7D47D54-745F-49A2-95F6-97E80B3A91D5 Q35723690-F470514C-EBCC-4F4F-87A1-A24089DB0BFB Q36330216-91652D70-0698-44E9-84D8-A246153B8137 Q36360621-D20F3CE0-2E82-4F20-BD2A-DD49A5E571CD Q36477231-255D14DB-BDBA-421E-8E9A-0D0F74841CEB Q36560380-25A7FED2-AB00-4DDE-86BB-C69CC4591F6B Q36909576-2325BB30-3A0E-4FF0-9FB0-489E993D211E Q36992673-A2AB0B09-2E03-47A3-AF10-AFBB186D293F Q37062734-8A4A4490-478B-4EFB-B6EC-D9B8CA22DEA8 Q37154169-74C94386-9130-49B1-A0EE-A4D3F5752093 Q37391748-A38C74ED-6BF4-44C4-9DC1-D954347436A8 Q37610600-AD83E5A0-E1A9-4610-AE33-23E7C5FF67CD Q37902133-CB1CE739-E9C0-422E-A5B6-73BB0D4E56AD Q38135224-0574E56B-26BA-43F8-B1D1-55A1BB7936FA Q38947274-EAD14D48-3D17-4159-BCE1-356E54D91F0B Q40041521-327C9E9C-9103-40B5-9489-50113B069B55 Q41640745-40BA67F4-CFA4-4C3E-B236-5551D1AD9983 Q41919807-C86BEE64-D4B5-4CC9-81AB-74E0BEE74A57 Q41930664-70CD7A05-503D-4401-9937-40AFE93722EB Q42770564-FAE89DD4-4A56-4E96-ACFE-919904A2C39F Q46231602-9B5B6392-DD6A-4091-B3E3-B3E73EF6692B Q46792282-1E07E152-5130-40AF-BF8E-C6B04F6140E2 Q46951093-12BEDB0A-8F5D-4037-8FF8-8B31AF6CAFB0 Q48795631-207747D1-09D7-4C90-BFF7-47A5D7DCE96D

P2860

Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathy.

http://www.wikidata.org/entity/Q34789824

description

2003 nî lūn-bûn

@nan

2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2003年の論文

@ja

2003年論文

@yue

2003年論文

@zh-hant

2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

@wuu

name

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@ast

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@en

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@nl

type

label

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@ast

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@en

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@nl

prefLabel

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@ast

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@en

Glucose transporter-1-deficien ...... milar to diabetic embryopathy.

@nl

P1433

Q34789824-7CB0BAAD-C3F6-4DBA-B334-AD8A5B1A33C8

P1476

Q34789824-D43EF74B-C3C8-4067-91B9-E2E2B04C8657

P2093

Q34789824-0C5D5A5F-89A7-4225-88D5-8DD12DF805CF

Q34789824-6B10BE84-5B85-4AF8-8397-7954A6C50FB9

Q34789824-703F4100-B7AA-429A-AF23-3AE972510505

Q34789824-76EA9B3F-436C-4B85-9533-FA58A064A047

Q34789824-9B23A972-FAED-4C84-8EDD-5952A45CEC78

Q34789824-AEECD6BB-8E50-4199-923E-CB1B06791B54

Q34789824-DEA7276C-6C3E-405E-B502-8EBC4060FB2F

P2860

Q34789824-114CC4E6-63FA-4A98-8FFF-CF39076FAAA0

Q34789824-26B9812A-A96E-46B2-8363-0642C67D7250

Q34789824-3FF6ADBB-E944-461A-A9D4-5AAE03298681

Q34789824-4D976C72-B1BB-49ED-A0A7-00AA4975CB55

Q34789824-5414A012-8618-4D97-9705-42F4CC3498A9

Q34789824-61DE7CA2-07EE-4EDA-973B-E2434318DDEB

Q34789824-63D3B46C-9EB9-42F7-B0B0-4E694EA01078

Q34789824-6F8CFF2A-8200-4314-9C1D-98F2C3D041A3

Q34789824-728EBC8C-B442-4D61-A46D-3EBC5C5F1A08

Q34789824-733A2174-5EF0-40A2-B59A-BEA00A7D48B1

P304

Q34789824-4208987D-F002-43D3-8A98-D7267F5891A2

P31

Q34789824-0AB9CCBF-ADDD-4C74-9F0E-2992F04DD3B3

P356

Q34789824-F8E9F9B5-5477-4805-975E-FE9D17307F17

P407

Q34789824-C500EA0B-D07C-400E-BC02-B850A93D96FB

P433

Q34789824-AD7C5E48-B2E9-4C25-BD69-857D81BB7375

P478

Q34789824-CD137180-5023-4BD8-AE71-0D1D78273389

P50

Q34789824-435EE45A-3B4A-4304-B235-DB1E438C47D1

P577

Q34789824-035C223E-6748-4A16-AB9F-A94DE8A1A3DA

P5875

Q34789824-C14216F4-B1D1-437D-8FF7-35ECF2C632C6

P698

Q34789824-461DACF7-ABBB-4D07-BD07-C120A64C6438

P932

Q34789824-0A8DA1A6-68D8-4047-9BBE-779119DE1575

P356

PNAS.2536196100

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Glucose transporter-1-deficien ...... imilar to diabetic embryopathy

@en

P2093

Charles W Heilig

http://www.w3.org/2001/XMLSchema#string

David Conner

http://www.w3.org/2001/XMLSchema#string

Frank C Brosius

http://www.w3.org/2001/XMLSchema#string

Kathleen Heilig

http://www.w3.org/2001/XMLSchema#string

Kelle Moley

http://www.w3.org/2001/XMLSchema#string

LiRong Guo

http://www.w3.org/2001/XMLSchema#string

Raymond Baggs

http://www.w3.org/2001/XMLSchema#string

P2860

GLUT8 is a glucose transporter responsible for insulin-stimulated glucose uptake in the blastocyst

Decreased glucose transporter expression triggers BAX-dependent apoptosis in the murine blastocyst

A human beta-actin expression vector system directs high-level accumulation of antisense transcripts.

Implications of glucose transporter protein type 1 (GLUT1)-haplodeficiency in embryonic stem cells for their survival in response to hypoxic stress.

The mammalian glucose transporters.

Transgenic mice overexpressing insulin-like growth factor-II in beta cells develop type 2 diabetes.

Antisense GLUT1 RNA suppresses the transforming phenotypes of NIH 3T3 cells transformed by N-Ras.

Antisense GLUT-1 protects mesangial cells from glucose induction of GLUT-1 and fibronectin expression.

Syntaxin 4 expression affects glucose transporter 8 translocation and embryo survival.

Perinatal outcome of pregnancies complicated by diabetes and by maternal daily hyperglycemia not related to diabetes. A retrospective 10-year analysis.

P304

15613-15618

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.2536196100

http://www.w3.org/2001/XMLSchema#string

P407

P433

26

http://www.w3.org/2001/XMLSchema#string

P478

100

http://www.w3.org/2001/XMLSchema#string

P50

Thomas L Saunders

P577

2003-12-12T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

8961750

http://www.w3.org/2001/XMLSchema#string

P698

14673082

http://www.w3.org/2001/XMLSchema#string

P932

307616

http://www.w3.org/2001/XMLSchema#string