Neuropathology of genetically engineered mice: consensus report and recommendations from an international forum. - Wikidata - wikimedia - TriplyDB

Neuropathology of genetically engineered mice: consensus report and recommendations from an international forum.

Neuropathology of genetically engineered mice: consensus report and recommendations from an international forum.

http://www.wikidata.org/entity/Q34974379

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Mouse models of cancer: Sleeping Beauty transposons for insertional mutagenesis screens and reverse genetic studies The Mouse Tumor Biology Database: integrated access to mouse cancer biology data.Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity.High-resolution longitudinal screening with magnetic resonance imaging in a murine brain cancer model.Pathological and molecular progression of astrocytomas in a GFAP:12 V-Ha-Ras mouse astrocytoma model.Mouse Tumor Biology (MTB): a database of mouse models for human cancer PTEN and NF1 inactivation in Schwann cells produces a severe phenotype in the peripheral nervous system that promotes the development and malignant progression of peripheral nerve sheath tumors.Conditional Inactivation of Pten with EGFR Overexpression in Schwann Cells Models Sporadic MPNST.Platelet-derived growth factor receptor alpha in glioma: a bad seed Modeling cancer patient populations in mice: complex genetic and environmental factors Rethinking brain tumors: the fourth Mouse Models of Human Cancers Consortium nervous system tumors workshop.De novo induction of genetically engineered brain tumors in mice using plasmid DNA Susceptibility to astrocytoma in mice mutant for Nf1 and Trp53 is linked to chromosome 11 and subject to epigenetic effects.Internet and print resources to facilitate pathology analysis when phenotyping genetically engineered rodents.Brain-specific 1B promoter of FGF1 gene facilitates the isolation of neural stem/progenitor cells with self-renewal and multipotent capacities.REST regulates oncogenic properties of glioblastoma stem cells.'One medicine---one pathology': are veterinary and human pathology prepared?Evolution of mouse models for studying CNS cancer: a decade of progress.Proliferative and nonproliferative lesions of the rat and mouse central and peripheral nervous systems.Cerebellar stem cells act as medulloblastoma-initiating cells in a mouse model and a neural stem cell signature characterizes a subset of human medulloblastomas.Human FGF1 promoter is active in ependymal cells and dopaminergic neurons in the brains of F1B-GFP transgenic mice.Bioluminescent approaches for measuring tumor growth in a mouse model of neurofibromatosis.

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Neuropathology of genetically engineered mice: consensus report and recommendations from an international forum.

http://www.wikidata.org/entity/Q34974379

description

2002 nî lūn-bûn

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2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2002年の論文

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Abhijit Guha

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Andrea I McClatchey

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C David James

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Charles Cobbs

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Cheryl Marks

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Ing-Ming Chiu

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Ivan Radovanovic

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P2860

The Nf2 tumor suppressor gene product is essential for extraembryonic development immediately prior to gastrulation

Ablation of NF1 function in neurons induces abnormal development of cerebral cortex and reactive gliosis in the brain

Neurofibromatosis 1 (NF1) heterozygosity results in a cell-autonomous growth advantage for astrocytes

Mice heterozygous for a mutation at the Nf2 tumor suppressor locus develop a range of highly metastatic tumors

Nf1;Trp53 mutant mice develop glioblastoma with evidence of strain-specific effects

Altered neural cell fates and medulloblastoma in mouse patched mutants

Mouse models of tumor development in neurofibromatosis type 1

Mouse tumor model for neurofibromatosis type 1

Region-specific astrogliosis in brains of mice heterozygous for mutations in the neurofibromatosis type 1 (Nf1) tumor suppressor

Tumour predisposition in mice heterozygous for a targeted mutation in Nf1

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