Role of transcription factor T-bet expression by CD4+ cells in gastritis due to Helicobacter pylori in mice - Wikidata - wikimedia - TriplyDB

Role of transcription factor T-bet expression by CD4+ cells in gastritis due to Helicobacter pylori in mice

Role of transcription factor T-bet expression by CD4+ cells in gastritis due to Helicobacter pylori in mice

http://www.wikidata.org/entity/Q34975829

about

Helicobacter pylori and T Helper Cells: Mechanisms of Immune Escape and Tolerance Antisense RNA modulation of alkyl hydroperoxide reductase levels in Helicobacter pylori correlates with organic peroxide toxicity but not infectivity.Systems modeling of the role of interleukin-21 in the maintenance of effector CD4+ T cell responses during chronic Helicobacter pylori infection.Th immune response induced by H pylori vaccine with chitosan as adjuvant and its relation to immune protection.Helicobacter pylori and gastric cancer: factors that modulate disease risk.Anti-inflammatory activity of bone morphogenetic protein signaling pathways in stomachs of mice Role of innate immunity in Helicobacter pylori-induced gastric malignancy.Ulcerative typhlocolitis associated with Helicobacter mastomyrinus in telomerase-deficient mice IFN-γ inhibits gastric carcinogenesis by inducing epithelial cell autophagy and T-cell apoptosis Immunization with dendritic cells pulsed ex vivo with recombinant chlamydial protease-like activity factor induces protective immunity against genital chlamydiamuridarum challenge Helicobacter pylori chemotaxis modulates inflammation and bacterium-gastric epithelium interactions in infected mice.The Helicobacter pylori autotransporter ImaA (HP0289) modulates the immune response and contributes to host colonization Innate-like CD4 T cells selected by thymocytes suppress adaptive immune responses against bacterial infections.Complex T cell interactions contribute to Helicobacter pylori gastritis in mice Impairment of ghrelin synthesis in Helicobacter pylori-colonized stomach: new clues for the pathogenesis of H. pylori-related gastric inflammation T-bet knockout prevents Helicobacter felis-induced gastric cancer.Regulation of Gastric Metaplasia, Dysplasia, and Neoplasia by Bone Morphogenetic Protein Signaling.Partial protection against Helicobacter pylori in the absence of mast cells in mice.Essential role for macrophage migration inhibitory factor in gastritis induced by Helicobacter pylori.Helicobacter pylori infection associates with a mucosal downregulation of ghrelin, negative regulator of Th1-cell responses.Interferon-γ-producing B cells induce the formation of gastric lymphoid follicles after Helicobacter suis infection.Regulation of Gastric Lgr5+ve Cell Homeostasis by Bone Morphogenetic Protein (BMP) Signaling and Inflammatory Stimuli.

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P2860

Role of transcription factor T-bet expression by CD4+ cells in gastritis due to Helicobacter pylori in mice

http://www.wikidata.org/entity/Q34975829

description

2006 nî lūn-bûn

@nan

2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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name

Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Role of transcription factor T ...... to Helicobacter pylori in mice

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Infection and Immunity

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Role of transcription factor T ...... to Helicobacter pylori in mice

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P2093

Brian M Gray

http://www.w3.org/2001/XMLSchema#string

Jennifer Haeger

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Kathryn A Eaton

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Lucy H Benson

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P2860

A novel transcription factor, T-bet, directs Th1 lineage commitment

Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

Sustained T-bet expression confers polarized human TH2 cells with TH1-like cytokine production and migratory capacities

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Divergent pro- and antiinflammatory roles for IL-23 and IL-12 in joint autoimmune inflammation

Gastric helicobacter infection induces a Th2 phenotype but does not elevate serum cholesterol in mice lacking inducible nitric oxide synthase.

Antrum- and corpus mucosa-infiltrating CD4(+) lymphocytes in Helicobacter pylori gastritis display a Th1 phenotype.

Campylobacter pylori: clinical, histological, and serological studies.

Role of gamma interferon in Helicobacter pylori-induced gastric inflammatory responses in a mouse model

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4673-4684

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scholarly article

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10.1128/IAI.01887-05

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8

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74

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2006-08-01T00:00:00Z

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16861655

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Helicobacter pylori

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1539619

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