Daily intermittent hypoxia augments spinal BDNF levels, ERK phosphorylation and respiratory long-term facilitation.
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Hypoxia-induced phrenic long-term facilitation: emergent propertiesUnexpected benefits of intermittent hypoxia: enhanced respiratory and nonrespiratory motor functionReactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic PlasticityNeuromechanical principles underlying movement modularity and their implications for rehabilitationThe effect of age and tongue exercise on BDNF and TrkB in the hypoglossal nucleus of ratsChronic intermittent hypoxia-induced deficits in synaptic plasticity and neurocognitive functions: a role for brain-derived neurotrophic factor.Differential expression of respiratory long-term facilitation among inbred rat strains.Preinspiratory and inspiratory hypoglossal motor output during hypoxia-induced plasticity in the rat.Chronic intermittent hypoxia affects integration of sensory input by neurons in the nucleus tractus solitariiRepetitive intermittent hypoxia induces respiratory and somatic motor recovery after chronic cervical spinal injuryDaily intermittent hypoxia enhances walking after chronic spinal cord injury: a randomized trial.Chronic intermittent hypoxia alters density of aminergic terminals and receptors in the hypoglossal motor nucleus.Remote limb ischemic conditioning enhances motor learning in healthy humans.Diaphragm long-term facilitation following acute intermittent hypoxia during wakefulness and sleep.Therapeutic potential of intermittent hypoxia: a matter of dose.Hypoglossal neuropathology and respiratory activity in pompe miceSpinal vascular endothelial growth factor induces phrenic motor facilitation via extracellular signal-regulated kinase and Akt signaling.Repetitive acute intermittent hypoxia increases expression of proteins associated with plasticity in the phrenic motor nucleusSimilarities and differences in mechanisms of phrenic and hypoglossal motor facilitation.Mammalian target of rapamycin is required for phrenic long-term facilitation following severe but not moderate acute intermittent hypoxia.Repetitive acute intermittent hypoxia does not promote generalized inflammatory gene expression in the rat CNS.Acute intermittent hypoxia induced phrenic long-term facilitation despite increased SOD1 expression in a rat model of ALS.Phrenic long-term facilitation after acute intermittent hypoxia requires spinal ERK activation but not TrkB synthesisRole of brain-derived neurotrophic factor in the excitatory-inhibitory imbalance during the critical period of postnatal respiratory development in the ratIntermittent hypoxia and neurorehabilitation.Cervical spinal erythropoietin induces phrenic motor facilitation via extracellular signal-regulated protein kinase and Akt signaling.Spinal atypical protein kinase C activity is necessary to stabilize inactivity-induced phrenic motor facilitation.Effect of acute intermittent hypoxia treatment on ventilatory load compensation and magnitude estimation of inspiratory resistive loads in an individual with chronic incomplete cervical spinal cord injuryTransporter Protein-Coupled DPCPX Nanoconjugates Induce Diaphragmatic Recovery after SCI by Blocking Adenosine A1 Receptors.Quantitative assessment of integrated phrenic nerve activity.Time Domains of the Hypoxic Ventilatory Response and Their Molecular Basis.Intermittent hypoxia induces functional recovery following cervical spinal injury.Repetitive acute intermittent hypoxia increases growth/neurotrophic factor expression in non-respiratory motor neuronsBDNF secretion by human pulmonary artery endothelial cells in response to hypoxia.Spinal plasticity following intermittent hypoxia: implications for spinal injury.Treatments to restore respiratory function after spinal cord injury and their implications for regeneration, plasticity and adaptation.Acute intermittent hypoxia induced neural plasticity in respiratory motor control.Spinal metaplasticity in respiratory motor controlEnhancing neural activity to drive respiratory plasticity following cervical spinal cord injury.The pattern and extent of retrograde transsynaptic transport of WGA-Alexa 488 in the phrenic motor system is dependent upon the site of application.
P2860
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P2860
Daily intermittent hypoxia augments spinal BDNF levels, ERK phosphorylation and respiratory long-term facilitation.
description
2009 nî lūn-bûn
@nan
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@ast
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@en
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@nl
type
label
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@ast
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@en
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@nl
prefLabel
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@ast
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@en
Daily intermittent hypoxia aug ...... ratory long-term facilitation.
@nl
P2860
P1476
Daily intermittent hypoxia aug ...... iratory long-term facilitation
@en
P2093
Gordon S Mitchell
P2860
P304
P356
10.1016/J.EXPNEUROL.2009.01.017
P407
P577
2009-02-03T00:00:00Z