Cervical spinal erythropoietin induces phrenic motor facilitation via extracellular signal-regulated protein kinase and Akt signaling.
about
Hypoxia-induced phrenic long-term facilitation: emergent propertiesUnexpected benefits of intermittent hypoxia: enhanced respiratory and nonrespiratory motor functionNeither serotonin nor adenosine-dependent mechanisms preserve ventilatory capacity in ALS ratsErythropoietin action in stress response, tissue maintenance and metabolism.Therapeutic potential of intermittent hypoxia: a matter of dose.Intermittent Hypoxia-Induced Spinal Inflammation Impairs Respiratory Motor Plasticity by a Spinal p38 MAP Kinase-Dependent MechanismMammalian target of rapamycin is required for phrenic long-term facilitation following severe but not moderate acute intermittent hypoxia.Acute intermittent hypoxia induced phrenic long-term facilitation despite increased SOD1 expression in a rat model of ALS.Phrenic long-term facilitation after acute intermittent hypoxia requires spinal ERK activation but not TrkB synthesisTime Domains of the Hypoxic Ventilatory Response and Their Molecular Basis.Intermittent hypoxia and stem cell implants preserve breathing capacity in a rodent model of amyotrophic lateral sclerosisRepeated intravenous doxapram induces phrenic motor facilitation.Common mechanisms of compensatory respiratory plasticity in spinal neurological disorders.Acute intermittent hypoxia induced neural plasticity in respiratory motor control.Axon plasticity in the mammalian central nervous system after injury.Spinal metaplasticity in respiratory motor controlPolycythemia and high levels of erythropoietin in blood and brain blunt the hypercapnic ventilatory response in adult mice.Signalling mechanisms of long term facilitation of breathing with intermittent hypoxia.Spinal vascular endothelial growth factor (VEGF) and erythropoietin (EPO) induced phrenic motor facilitation after repetitive acute intermittent hypoxia.Mechanisms of Enhanced Phrenic Long-Term Facilitation in SOD1G93A Rats.Cyclooxygenase enzyme activity does not impair respiratory motor plasticity after one night of intermittent hypoxia.Enhancement of phrenic long-term facilitation following repetitive acute intermittent hypoxia is blocked by the glycolytic inhibitor 2-deoxyglucose.Spinal BDNF-induced phrenic motor facilitation requires PKCθ activity.Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.Acute intermittent hypoxia and rehabilitative training following cervical spinal injury alters neuronal hypoxia- and plasticity-associated protein expression.
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P2860
Cervical spinal erythropoietin induces phrenic motor facilitation via extracellular signal-regulated protein kinase and Akt signaling.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@ast
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@en
type
label
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@ast
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@en
prefLabel
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@ast
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@en
P2093
P2860
P1476
Cervical spinal erythropoietin ...... tein kinase and Akt signaling.
@en
P2093
Erica A Dale
Gordon S Mitchell
Irawan Satriotomo
P2860
P304
P356
10.1523/JNEUROSCI.3873-11.2012
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P577
2012-04-01T00:00:00Z