Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage. - Wikidata - wikimedia - TriplyDB

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

http://www.wikidata.org/entity/Q34987124

about

Variant base excision repair proteins: contributors to genomic instability DNA repair targeted therapy: The past or future of cancer treatment?Hsp90: A New Player in DNA Repair?The maintenance of mitochondrial DNA integrity--critical analysis and update Introduction to radiobiology of targeted radionuclide therapy Mitochondrial DNA damage and its consequences for mitochondrial gene expression Repair of persistent strand breaks in the mitochondrial genome DNA methylation, its mediators and genome integrity Genetic instability in budding and fission yeast-sources and mechanisms Base excision repair facilitates a functional relationship between Guanine oxidation and histone demethylation Base excision repair Concomitant inactivation of foxo3a and fancc or fancd2 reveals a two-tier protection from oxidative stress-induced hydrocephalus Guanine holes are prominent targets for mutation in cancer and inherited disease Single nucleotide polymorphisms as susceptibility, prognostic, and therapeutic markers of nonsmall cell lung cancer WRN is recruited to damaged telomeres via its RQC domain and tankyrase1-mediated poly-ADP-ribosylation of TRF1.XRCC1 Arg399Gln and Arg194Trp polymorphisms and risk of systemic lupus erythematosus in an Iranian population: a pilot study Base excision repair: a critical player in many games Discordant in vitro and in vivo chemopotentiating effects of the PARP inhibitor veliparib in temozolomide-sensitive versus -resistant glioblastoma multiforme xenografts.ARTD1/PARP1 negatively regulates glycolysis by inhibiting hexokinase 1 independent of NAD+ depletion.DNA mismatch repair and oxidative DNA damage: implications for cancer biology and treatment.Mitochondrial DNA ligase is dispensable for the viability of cultured cells but essential for mtDNA maintenance.Alkylation sensitivity screens reveal a conserved cross-species functionome.Aag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney.Impact of age-associated cyclopurine lesions on DNA repair helicases.HSP90 regulates DNA repair via the interaction between XRCC1 and DNA polymerase β Base excision repair capacity in informing healthspan.Ataxia telangiectasia mutated and Rad3 related (ATR) protein kinase inhibition is synthetically lethal in XRCC1 deficient ovarian cancer cells.The emerging nexus of active DNA demethylation and mitochondrial oxidative metabolism in post-mitotic neurons Defective repair of oxidative base lesions by the DNA glycosylase Nth1 associates with multiple telomere defects.Identification and quantification of DNA repair protein apurinic/apyrimidinic endonuclease 1 (APE1) in human cells by liquid chromatography/isotope-dilution tandem mass spectrometry.N-methylpurine DNA glycosylase and DNA polymerase beta modulate BER inhibitor potentiation of glioma cells to temozolomide Interplay between DNA repair and inflammation, and the link to cancer DNA damage and autophagy.Novel method for site-specific induction of oxidative DNA damage reveals differences in recruitment of repair proteins to heterochromatin and euchromatin Repair of oxidative DNA base damage in the host genome influences the HIV integration site sequence preference.Functional analyses of human DNA repair proteins important for aging and genomic stability using yeast genetics The FA pathway counteracts oxidative stress through selective protection of antioxidant defense gene promoters Salidroside stimulates DNA repair enzyme Parp-1 activity in mouse HSC maintenance.Colon cancer-associated DNA polymerase β variant induces genomic instability and cellular transformation.RECQ1 plays a distinct role in cellular response to oxidative DNA damage.

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P2860

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

http://www.wikidata.org/entity/Q34987124

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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type

label

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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prefLabel

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

@en

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

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Antioxidants & Redox Signaling

P1476

Base excision repair and lesion-dependent subpathways for repair of oxidative DNA damage.

@en

P2093

David Svilar

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Eva M Goellner

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Karen H Almeida

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Robert W Sobol

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P2860

The nuclear DNA base 5-hydroxymethylcytosine is present in Purkinje neurons and the brain

DNA Damage, Aging, and Cancer

Definitive identification of mammalian 5-hydroxymethyluracil DNA N-glycosylase activity as SMUG1

Mammalian 5-formyluracil-DNA glycosylase. 2. Role of SMUG1 uracil-DNA glycosylase in repair of 5-formyluracil and other oxidized and deaminated base lesions

Ku is a 5'-dRP/AP lyase that excises nucleotide damage near broken ends

Human DNA2 is a mitochondrial nuclease/helicase for efficient processing of DNA replication and repair intermediates

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

Acetylation of the human DNA glycosylase NEIL2 and inhibition of its activity

Escherichia coli Nth and human hNTH1 DNA glycosylases are involved in removal of 8-oxoguanine from 8-oxoguanine/guanine mispairs in DNA

NEIL2-initiated, APE-independent repair of oxidized bases in DNA: Evidence for a repair complex in human cells

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2491-2507

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scholarly article

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10.1089/ARS.2010.3466

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12

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14

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45280542

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20649466

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3096496

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