Reversal of West Nile virus-induced blood-brain barrier disruption and tight junction proteins degradation by matrix metalloproteinases inhibitor.
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Mechanism of West Nile virus neuroinvasion: a critical appraisalIlluminating viral infections in the nervous systemIn Vitro Infection with Dengue Virus Induces Changes in the Structure and Function of the Mouse Brain EndotheliumInnate immune interactions within the central nervous system modulate pathogenesis of viral infectionsInflammasome adaptor protein Apoptosis-associated speck-like protein containing CARD (ASC) is critical for the immune response and survival in west Nile virus encephalitisIn Vitro and In Vivo Blood-Brain Barrier Models to Study West Nile Virus Pathogenesis.West Nile virus-induced cell adhesion molecules on human brain microvascular endothelial cells regulate leukocyte adhesion and modulate permeability of the in vitro blood-brain barrier modelStructural proteins of West Nile virus are a major determinant of infectious particle production and fitness in astrocytes.Viral pathogen-associated molecular patterns regulate blood-brain barrier integrity via competing innate cytokine signals.2'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivoWest Nile virus infection causes endocytosis of a specific subset of tight junction membrane proteins.Pro-inflammatory cytokines derived from West Nile virus (WNV)-infected SK-N-SH cells mediate neuroinflammatory markers and neuronal deathIL-22 signaling contributes to West Nile encephalitis pathogenesis.Combined treatment of adenosine nucleoside inhibitor NITD008 and histone deacetylase inhibitor vorinostat represents an immunotherapy strategy to ameliorate West Nile virus infection.VEGFR2 and Src kinase inhibitors suppress Andes virus-induced endothelial cell permeabilityIgG dynamics of dietary antigens point to cerebrospinal fluid barrier or flow dysfunction in first-episode schizophrenia.Cyclooxygenase-2 inhibitor blocks the production of West Nile virus-induced neuroinflammatory markers in astrocytes.Elevation of matrix metalloproteinase-9 level in cerebrospinal fluid of tick-borne encephalitis patients is associated with IgG extravassation and disease severity.West Nile virus: immunity and pathogenesis.Viral disruption of the blood-brain barrier.Contribution of a single host genetic locus to mouse adenovirus type 1 infection and encephalitis.Induction of virus-specific effector immune cell response limits virus replication and severe disease in mice infected with non-lethal West Nile virus Eg101 strain.Viral interactions with the blood-brain barrier: old dog, new tricksVirus infections in the nervous systemJapanese encephalitis virus disrupts cell-cell junctions and affects the epithelial permeability barrier functionsWest Nile virus-induced disruption of the blood-brain barrier in mice is characterized by the degradation of the junctional complex proteins and increase in multiple matrix metalloproteinases.Encephalitic Arboviruses: Emergence, Clinical Presentation, and Neuropathogenesis.Death receptor-mediated apoptotic signaling is activated in the brain following infection with West Nile virus in the absence of a peripheral immune response.Infection of pericytes in vitro by Japanese encephalitis virus disrupts the integrity of the endothelial barrier.Targeting host factors to treat West Nile and dengue viral infections.Differential induction of CCL5 by pathogenic and non-pathogenic strains of West Nile virus in brain endothelial cells and astrocytes.Reduced immune cell infiltration and increased pro-inflammatory mediators in the brain of Type 2 diabetic mouse model infected with West Nile virus.Occludin: one protein, many forms.Immune responses to West Nile virus infection in the central nervous system.West Nile virus infection and immunity.Claudins and other tight junction proteins.The neuroimmune response to West Nile virus.Innate host responses to West Nile virus: Implications for central nervous system immunopathology.MMP-9 in translation: from molecule to brain physiology, pathology, and therapy.Mechanisms of restriction of viral neuroinvasion at the blood-brain barrier.
P2860
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P2860
Reversal of West Nile virus-induced blood-brain barrier disruption and tight junction proteins degradation by matrix metalloproteinases inhibitor.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@ast
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@en
type
label
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@ast
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@en
prefLabel
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@ast
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@en
P2093
P2860
P1433
P1476
Reversal of West Nile virus-in ...... metalloproteinases inhibitor.
@en
P2093
Saguna Verma
Stephanie Lum
Ulziijargal Gurjav
Vivek R Nerurkar
P2860
P304
P356
10.1016/J.VIROL.2009.10.036
P407
P50
P577
2009-11-18T00:00:00Z