TNF-alpha downregulates eNOS expression and mitochondrial biogenesis in fat and muscle of obese rodents. - Wikidata - wikimedia - TriplyDB

TNF-alpha downregulates eNOS expression and mitochondrial biogenesis in fat and muscle of obese rodents.

TNF-alpha downregulates eNOS expression and mitochondrial biogenesis in fat and muscle of obese rodents.

http://www.wikidata.org/entity/Q35025561

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Neuroinflammation and psychiatric illness Role of Energy Metabolism in the Brown Fat Gene Program Endothelial dysfunction and preeclampsia: role of oxidative stress Mitochondrial dysfunction in obesity: potential benefit and mechanism of Co-enzyme Q10 supplementation in metabolic syndrome Free fatty acids, lipopolysaccharide and IL-1α induce adipocyte manganese superoxide dismutase which is increased in visceral adipose tissues of obese rodents Oxidative stress, cardiolipin and mitochondrial dysfunction in nonalcoholic fatty liver disease Global transcript profiles of fat in monozygotic twins discordant for BMI: pathways behind acquired obesity Tumor necrosis factor α decreases nitric oxide synthase type 3 expression primarily via Rho/Rho kinase in the thick ascending limb Exercise and adrenaline increase PGC-1{alpha} mRNA expression in rat adipose tissue Sustained hypoxia modulates mitochondrial DNA content in the neonatal rat brain Genetic and epigenetic factors are associated with expression of respiratory chain component NDUFB6 in human skeletal muscle Regulation of obesity and insulin resistance by nitric oxide Effects of diet-induced obesity on protein expression in insulin signaling pathways of skeletal muscle in male Wistar rats.Role of the immune system in hypertension: modulation by dietary antioxidants.Monomeric tartrate resistant acid phosphatase induces insulin sensitive obesity Cycling our way to fit fat.Proinflammatory cytokines differentially regulate adipocyte mitochondrial metabolism, oxidative stress, and dynamics.Polycystic Ovary Syndrome: Insights into the Therapeutic Approach with Inositols.The association of insertions/deletions (INDELs) and variable number tandem repeats (VNTRs) with obesity and its related traits and complications Combined effects of ghrelin and higher food intake enhance skeletal muscle mitochondrial oxidative capacity and AKT phosphorylation in rats with chronic kidney disease.Downregulation of adipose glutathione S-transferase A4 leads to increased protein carbonylation, oxidative stress, and mitochondrial dysfunction.Endothelial nitric oxide synthase is central to skeletal muscle metabolic regulation and enzymatic signaling during exercise in vivo.Effects of dipeptidyl-peptidase 4 inhibitor about vascular inflammation in a metabolic syndrome model.Impaired mitochondrial function in human placenta with increased maternal adiposity Unraveling biochemical pathways affected by mitochondrial dysfunctions using metabolomic approaches.GIP-overexpressing mice demonstrate reduced diet-induced obesity and steatosis, and improved glucose homeostasis IL-6 is not necessary for the regulation of adipose tissue mitochondrial content.Impact of nutrient excess and endothelial nitric oxide synthase on the plasma metabolite profile in mice.Corepressor SMRT promotes oxidative phosphorylation in adipose tissue and protects against diet-induced obesity and insulin resistance.Respiratory and skeletal muscle strength in chronic obstructive pulmonary disease: impact on exercise capacity and lower extremity function.Quercetin protects against obesity-induced skeletal muscle inflammation and atrophy.Nitric oxide donors rescue diabetic nephropathy through oxidative-stress-and nitrosative-stress-mediated Wnt signaling pathways.Nitric oxide, interorganelle communication, and energy flow: a novel route to slow aging.Adenosine prevents TNFα-induced decrease in endothelial mitochondrial mass via activation of eNOS-PGC-1α regulatory axis.Metabolic syndrome and obesity in an insect.Antiobesogenic role of endothelial nitric oxide synthase.Alterations of cellular bioenergetics in pulmonary artery endothelial cells Chronic deficiency of nitric oxide affects hypoxia inducible factor-1α (HIF-1α) stability and migration in human endothelial cells.The non-invasive (13)C-methionine breath test detects hepatic mitochondrial dysfunction as a marker of disease activity in non-alcoholic steatohepatitis Limited OXPHOS capacity in white adipocytes is a hallmark of obesity in laboratory mice irrespective of the glucose tolerance status.

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P2860

TNF-alpha downregulates eNOS expression and mitochondrial biogenesis in fat and muscle of obese rodents.

http://www.wikidata.org/entity/Q35025561

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2006 nî lūn-bûn

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2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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TNF-alpha downregulates eNOS e ...... t and muscle of obese rodents.

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Addolorata Pisconti

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Annalisa Cardile

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Laura Tedesco

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Letizia Palomba

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Michele O Carruba

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Orazio Cantoni

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Salvador Moncada

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Valeria Cozzi

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P2860

Inflammation, stress, and diabetes

Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

Coordinated reduction of genes of oxidative metabolism in humans with insulin resistance and diabetes: Potential role of PGC1 and NRF1

Obesity is associated with macrophage accumulation in adipose tissue

Cloning and mRNA tissue distribution of human PPARgamma coactivator-1.

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

Cellular and molecular regulation of muscle regeneration

Mitofusin-2 determines mitochondrial network architecture and mitochondrial metabolism. A novel regulatory mechanism altered in obesity

Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance

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