Pathogenesis of indirect (secondary) acute lung injury. - Wikidata - wikimedia - TriplyDB

Pathogenesis of indirect (secondary) acute lung injury.

Pathogenesis of indirect (secondary) acute lung injury.

http://www.wikidata.org/entity/Q35026951

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Delivery of RNAi Therapeutics to the Airways-From Bench to Bedside Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrity TAT-SNAP-23 treatment inhibits the priming of neutrophil functions contributing to shock and/or sepsis-induced extra-pulmonary acute lung injury The biological effects of higher and lower positive end-expiratory pressure in pulmonary and extrapulmonary acute lung injury with intra-abdominal hypertension Active players in resolution of shock/sepsis induced indirect lung injury: immunomodulatory effects of Tregs and PD-1.Human alveolar epithelial cells attenuate pulmonary microvascular endothelial cell permeability under septic conditions.The ratio of Th17/Treg cells as a risk indicator in early acute respiratory distress syndrome TAT-HSP70 Attenuates Experimental Lung Injury Biodistribution and Efficacy of Targeted Pulmonary Delivery of a Protein Kinase C-δ Inhibitory Peptide: Impact on Indirect Lung Injury.Isoflurane prevents acute lung injury through ADP-mediated platelet inhibition Blockage of thymic stromal lymphopoietin signaling improves acute lung injury in mice by regulating pulmonary dendritic cells Imbalance of Th17/Tregs in rats with smoke inhalation-induced acute lung injury.Glycyrrhizic Acid Prevents Sepsis-Induced Acute Lung Injury and Mortality in Rats.Protective effect of Astragaloside IV against sepsis-induced acute lung injury in rats.Pulmonary endothelial protein kinase C-delta (PKCδ) regulates neutrophil migration in acute lung inflammation.Sepsis: multiple abnormalities, heterogeneous responses, and evolving understanding.Pneumonia, Acute Respiratory Distress Syndrome, and Early Immune-Modulator Therapy.Acute lung injury results from failure of neutrophil de-priming: a new hypothesis.Endothelial progenitor cells: the promise of cell-based therapies for acute lung injury.Paracrine factors from mesenchymal stem cells: a proposed therapeutic tool for acute lung injury and acute respiratory distress syndrome.Intersectin-1s: an important regulator of cellular and molecular pathways in lung injury.Clinical and biological heterogeneity in acute respiratory distress syndrome: direct versus indirect lung injury.Metabotyping Patients' Journeys Reveals Early Predisposition to Lung Injury after Cardiac Surgery.Myocardial ischemic post-conditioning protects the lung against myocardial ischemia/reperfusion-induced damage by activating GSK-3β.Possible role for anisodamine in organophosphate poisoning.Remote Burn Injury Increases Pulmonary Histone Deacetylase 1 and Reduces Histone Acetylation.Early induction of uncoupling protein-2 in pulmonary macrophages in hyperoxia-associated lung injury.Characterization of blunt chest trauma in a long-term porcine model of severe multiple trauma.Clinical Predictors of Hospital Mortality Differ Between Direct and Indirect ARDS.Role of pulmonary microvascular endothelial cell apoptosis in murine sepsis-induced lung injury in vivo.Effects of Zinc Finger Protein A20 on Lipopolysaccharide (LPS)-Induced Pulmonary Inflammation/Anti-Inflammatory Mediators in an Acute Lung Injury/Acute Respiratory Distress Syndrome Rat Model.The anesthetic agent sevoflurane attenuates pulmonary acute lung injury by modulating apoptotic pathways.Pulmonary Endothelial Cell Apoptosis in Emphysema and Acute Lung Injury.PAD4 Deficiency Leads to Decreased Organ Dysfunction and Improved Survival in a Dual Insult Model of Hemorrhagic Shock and Sepsis.Therapeutic blockade of CD54 attenuates pulmonary barrier damage in T cell-induced acute lung injury.IL-27 is elevated in acute lung injury and mediates inflammation.Immunothrombotic Activity of Damage-Associated Molecular Patterns and Extracellular Vesicles in Secondary Organ Failure Induced by Trauma and Sterile Insults.Effects of SDF-1/CXCR4 on Acute Lung Injury Induced by Cardiopulmonary Bypass.Predictive biomarkers and metabolic hallmark of postoperative hypoxaemia

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Pathogenesis of indirect (secondary) acute lung injury.

http://www.wikidata.org/entity/Q35026951

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

@zh-hans

2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Pathogenesis of indirect (secondary) acute lung injury.

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Pathogenesis of indirect (secondary) acute lung injury.

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Pathogenesis of indirect (secondary) acute lung injury.

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Pathogenesis of indirect (secondary) acute lung injury.

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Pathogenesis of indirect (secondary) acute lung injury.

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Pathogenesis of indirect (secondary) acute lung injury.

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Expert Review of Respiratory Medicine

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Pathogenesis of indirect (secondary) acute lung injury.

@en

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Chun-Shiang Chung

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Fabienne Venet

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Joanne Lomas-Neira

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Mario Perl

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The aquaporin water channels

Fas and fas ligand are up-regulated in pulmonary edema fluid and lung tissue of patients with acute lung injury and the acute respiratory distress syndrome

Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. The Acute Respiratory Distress Syndrome Network

Resolution of lung inflammation by CD44

Impaired immunity and enhanced resistance to endotoxin in the absence of neutrophil elastase and cathepsin G

Incidence and outcomes of acute lung injury

The acute respiratory distress syndrome

The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination

Filgrastim in patients with pneumonia and severe sepsis or septic shock.

CESAR: conventional ventilatory support vs extracorporeal membrane oxygenation for severe adult respiratory failure

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115-126

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scholarly article

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10.1586/ERS.10.92

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