Angiotensin II induced catabolic effect and muscle atrophy are redox dependent. - Wikidata - wikimedia - TriplyDB

Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

http://www.wikidata.org/entity/Q35028013

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Aerobic Exercise and Pharmacological Therapies for Skeletal Myopathy in Heart Failure: Similarities and Differences Mitochondrial dysfunction in heart failure Salt-Sensitive Hypertension: Perspectives on Intrarenal Mechanisms Reactive Oxygen Species/Nitric Oxide Mediated Inter-Organ Communication in Skeletal Muscle Wasting Diseases.Effects of exercise training on circulating and skeletal muscle renin-angiotensin system in chronic heart failure rats Crossroads between peripheral atherosclerosis, western-type diet and skeletal muscle pathophysiology: emphasis on apolipoprotein E deficiency and peripheral arterial disease.Angiotensin II infusion induces marked diaphragmatic skeletal muscle atrophy Angiotensin type 2 receptor signaling in satellite cells potentiates skeletal muscle regeneration.Diaphragm dysfunction caused by sphingomyelinase requires the p47(phox) subunit of NADPH oxidase.Bortezomib, a proteasome inhibitor, attenuates angiotensin II-induced hypertension and aortic remodeling in rats.Angiotensin II upregulates protein phosphatase 2Cα and inhibits AMP-activated protein kinase signaling and energy balance leading to skeletal muscle wasting Effects of exercise training on neurovascular control and skeletal myopathy in systolic heart failure.AT1 receptor-mediated augmentation of angiotensinogen, oxidative stress, and inflammation in ANG II-salt hypertension.Angiotensin II reduces food intake by altering orexigenic neuropeptide expression in the mouse hypothalamus.Losartan treatment attenuates tumor-induced myocardial dysfunction NAD(P)H oxidase subunit p47phox is elevated, and p47phox knockout prevents diaphragm contractile dysfunction in heart failure.Mechanisms of Cachexia in Chronic Disease States Short-term nonpressor angiotensin II infusion stimulates sodium transporters in proximal tubule and distal nephron.AT1 receptor blocker losartan protects against mechanical ventilation-induced diaphragmatic dysfunction Angiotensin II inhibits satellite cell proliferation and prevents skeletal muscle regeneration.Molecular mechanisms and signaling pathways of angiotensin II-induced muscle wasting: potential therapeutic targets for cardiac cachexia Differential regulation of Na+ transporters along nephron during ANG II-dependent hypertension: distal stimulation counteracted by proximal inhibition Renin-angiotensin system: an old player with novel functions in skeletal muscle.The heritable path of human physical performance: from single polymorphisms to the "next generation".Enalapril treatment discloses an early role of angiotensin II in inflammation- and oxidative stress-related muscle damage in dystrophic mdx mice Regulation of NADPH oxidases in skeletal muscle.Angiotensin-(1-7) decreases skeletal muscle atrophy induced by angiotensin II through a Mas receptor-dependent mechanism.THE RENIN-ANGIOTENSIN SYSTEM AND THE BIOLOGY OF SKELETAL MUSCLE: MECHANISMS OF MUSCLE WASTING IN CHRONIC DISEASE STATES.Indoxyl sulfate potentiates skeletal muscle atrophy by inducing the oxidative stress-mediated expression of myostatin and atrogin-1.Detrimental Effects of Centrally Administered Angiotensin II are Enhanced in a Mouse Model of Alzheimer Disease Independently of Blood Pressure.Angiotensin II Induces Skeletal Muscle Atrophy by Activating TFEB-Mediated MuRF1 Expression.An Intronic Enhancer Element Regulates Angiotensin II Type 2 Receptor Expression during Satellite Cell Differentiation, and Its Activity Is Suppressed in Congestive Heart Failure.Diaphragm abnormalities in heart failure and aging: mechanisms and integration of cardiovascular and respiratory pathophysiology.The angiotensin-(1-7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice.Expression of the Mas receptor is upregulated in skeletal muscle wasting.Skeletal muscle bioenergetics in aging and heart failure.Possible Role of NADPH Oxidase 4 in Angiotensin II-Induced Muscle Wasting in Mice.Deletion of NAD(P)H Oxidase 2 Prevents Angiotensin II-Induced Skeletal Muscle Atrophy.Role of Oxidative Stress as Key Regulator of Muscle Wasting during Cachexia.Benzbromarone Attenuates Oxidative Stress in Angiotensin II- and Salt-Induced Hypertensive Model Rats.

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Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

http://www.wikidata.org/entity/Q35028013

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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type

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Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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prefLabel

Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

@ast

Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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J.BBRC.2011.04.122

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Biochemical and Biophysical Research Communications

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Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.

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P2093

A Michael Tabony

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Bashir M Rezk

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Charlotte Vaughn

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Laura C Semprun-Prieto

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Romer A Gonzalez-Villalobos

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P2860

Homologs of gp91phox: cloning and tissue expression of Nox3, Nox4, and Nox5

H(2)O(2)-induced O(2) production by a non-phagocytic NAD(P)H oxidase causes oxidant injury

Angiotensin II stimulates endothelial vascular cell adhesion molecule-1 via nuclear factor-kappaB activation induced by intracellular oxidative stress

Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy

NAD(P)H oxidase: role in cardiovascular biology and disease

Mechanisms underlying endothelial dysfunction in diabetes mellitus.

The effects of enalapril-digoxin-diuretic combination therapy on nutritional and anthropometric indices in chronic congestive heart failure: preliminary findings in cardiac cachexia.

Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

Role of superoxide in angiotensin II-induced but not catecholamine-induced hypertension.

Cardiac cachexia.

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217-221

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scholarly article

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10.1016/J.BBRC.2011.04.122

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2

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Patrice Delafontaine

Tadashi Yoshida

Sergiy Sukhanov

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2011-05-06T00:00:00Z

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51125512

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21570954

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