Deletion polymorphism of Disc1 is common to all 129 mouse substrains: implications for gene-targeting studies of brain function.
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Density and function of central serotonin (5-HT) transporters, 5-HT1A and 5-HT2A receptors, and effects of their targeting on BTBR T+tf/J mouse social behaviorSynaptic dysfunction and abnormal behaviors in mice lacking major isoforms of Shank3The DISC locus in psychiatric illnessDISC1 regulates new neuron development in the adult brain via modulation of AKT-mTOR signaling through KIAA1212Effects of NK-4 in a transgenic mouse model of Alzheimer's diseaseStrain differences in developmental vulnerability to alcohol exposure via embryo culture in mice.Mice with a naturally occurring DISC1 mutation display a broad spectrum of behaviors associated to psychiatric disorders.Contribution of nonprimate animal models in understanding the etiology of schizophrenia.Modeling the positive symptoms of schizophrenia in genetically modified mice: pharmacology and methodology aspectsThe nucleosome assembly protein TSPYL2 regulates the expression of NMDA receptor subunits GluN2A and GluN2B.The natural Disc1-deletion present in several inbred mouse strains does not affect sleep.Parallel universes of Black Six biology.Migration defects by DISC1 knockdown in C57BL/6, 129X1/SvJ, and ICR strains via in utero gene transfer and virus-mediated RNAi.The BTBR mouse model of autism spectrum disorders has learning and attentional impairments and alterations in acetylcholine and kynurenic acid in prefrontal cortex.Association of brain immune genes with social behavior of inbred mouse strains.Functional themes from psychiatric genome-wide screens.Agenesis of corpus callosum and emotional information processing in schizophrenia.The telomeric part of the human chromosome 21 from Cstb to Prmt2 is not necessary for the locomotor and short-term memory deficits observed in the Tc1 mouse model of Down syndromeMaternal immune activation and strain specific interactions in the development of autism-like behaviors in mice.Disrupted in schizophrenia 1 and phosphodiesterase 4B: towards an understanding of psychiatric illness.Dissociation of frontotemporal dementia-related deficits and neuroinflammation in progranulin haploinsufficient mice.Neurodevelopmental mechanisms of schizophrenia: understanding disturbed postnatal brain maturation through neuregulin-1-ErbB4 and DISC1.The audacity of proteomics: a chance to overcome current challenges in schizophrenia research.The gene or not the gene--that is the question: understanding the genetically engineered mouse phenotype.Modeling autism by SHANK gene mutations in mice.Disrupted in schizophrenia 1 and synaptic function in the mammalian central nervous system.Fiber geometry in the corpus callosum in schizophrenia: evidence for transcallosal misconnection.Phenotypic characterization of C57BL/6J mice carrying the Disc1 gene from the 129S6/SvEv strain.Malformation of the Posterior Cerebellar Vermis Is a Common Neuroanatomical Phenotype of Genetically Engineered Mice on the C57BL/6 Background.Deficiency of Shank2 causes mania-like behavior that responds to mood stabilizers.Disrupted-in-schizophrenia 1 regulates transport of ITPR1 mRNA for synaptic plasticity.Behavioral alterations associated with targeted disruption of exons 2 and 3 of the Disc1 gene in the mouse.Disc1 deletion is present in Swiss-derived inbred mouse strains: implications for transgenic studies of learning and memory.Minimizing strain influences in a genetically modified mouse phenotyping platform.Repeated Administration of D-Amphetamine Induces Distinct Alterations in Behavior and Metabolite Levels in 129Sv and Bl6 Mouse Strains.
P2860
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P2860
Deletion polymorphism of Disc1 is common to all 129 mouse substrains: implications for gene-targeting studies of brain function.
description
2006 nî lūn-bûn
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2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
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2006 թվականի հունիսին հրատարակված գիտական հոդված
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2006年の論文
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2006年論文
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2006年論文
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2006年論文
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2006年論文
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2006年論文
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2006年论文
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name
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@ast
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@en
type
label
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@ast
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@en
prefLabel
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@ast
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@en
P2860
P1433
P1476
Deletion polymorphism of Disc1 ...... ing studies of brain function.
@en
P2093
John C Roder
Steven J Clapcote
P2860
P304
P356
10.1534/GENETICS.106.060749
P407
P577
2006-06-04T00:00:00Z