Prenatal cocaine exposure increases apoptosis of neonatal rat heart and heart susceptibility to ischemia-reperfusion injury in 1-month-old rat.
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Fetal stress and programming of hypoxic/ischemic-sensitive phenotype in the neonatal brain: mechanisms and possible interventionsCocaine exposure in vitro induces apoptosis in fetal locus coeruleus neurons by altering the Bax/Bcl-2 ratio and through caspase-3 apoptotic signalingMaternal hypoxia increases the activity of MMPs and decreases the expression of TIMPs in the brain of neonatal ratsSystemic maternal inflammation and neonatal hyperoxia induces remodeling and left ventricular dysfunction in miceEnvironmental alterations of epigenetics prior to the birth.Postnatal consequences of prenatal cocaine exposure and myocardial apoptosis: does cocaine in utero imperil the adult heart?In utero exposure to diesel exhaust air pollution promotes adverse intrauterine conditions, resulting in weight gain, altered blood pressure, and increased susceptibility to heart failure in adult mice.Protective Effect of Antenatal Antioxidant on Nicotine-Induced Heart Ischemia-Sensitive Phenotype in Rat OffspringMinocycline suppresses oxidative stress and attenuates fetal cardiac myocyte apoptosis triggered by in utero cocaine exposure.Prenatal nicotine exposure increases heart susceptibility to ischemia/reperfusion injury in adult offspring.Fetal programming of cardiac function and disease.Prenatal cocaine exposure abolished ischemic preconditioning-induced protection in adult male rat hearts: role of PKCepsilon.Fetal exposure to cocaine causes programming of Prkce gene repression in the left ventricle of adult rat offspring.Short- and long-term adverse effects of cocaine abuse during pregnancy on the heart development.Direct effect of cocaine on epigenetic regulation of PKCepsilon gene repression in the fetal rat heart.Long-term health consequences of early-life exposure to substance abuse: an epigenetic perspective.Prenatal methamphetamine differentially alters myocardial sensitivity to ischemic injury in male and female adult hearts.Myocardial oxidative damage is induced by cardiac Fas-dependent and mitochondria-dependent apoptotic pathways in human cocaine-related overdosePrenatal cocaine exposure increases heart susceptibility to ischaemia-reperfusion injury in adult male but not female rats.
P2860
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P2860
Prenatal cocaine exposure increases apoptosis of neonatal rat heart and heart susceptibility to ischemia-reperfusion injury in 1-month-old rat.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@ast
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@en
type
label
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@ast
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@en
prefLabel
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@ast
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@en
P2860
P356
P1476
Prenatal cocaine exposure incr ...... ion injury in 1-month-old rat.
@en
P2093
Lubo Zhang
Soochan Bae
P2860
P304
P356
10.1038/SJ.BJP.0706129
P407
P577
2005-04-01T00:00:00Z