about
Murine muscle cell models for Pompe disease and their use in studying therapeutic approachesActin Filaments Are Involved in the Coupling of V0-V1 Domains of Vacuolar H+-ATPase at the Golgi Complex.Genome-wide analysis reveals the vacuolar pH-stat of Saccharomyces cerevisiae.Functional vacuolar ATPase (V-ATPase) proton pumps traffic to the enterocyte brush border membrane and require CFTR.The pH of the secretory pathway: measurement, determinants, and regulation.Elevated Golgi pH in breast and colorectal cancer cells correlates with the expression of oncofetal carbohydrate T-antigen.Sequestration of AS-DACA into acidic compartments of the membrane trafficking system as a mechanism of drug resistance in rhabdomyosarcomaInsights from bacterial subtilases into the mechanisms of intramolecular chaperone-mediated activation of furinRecombinant antibody mediated delivery of organelle-specific DNA pH sensors along endocytic pathways.NHE8 is essential for RPE cell polarity and photoreceptor survival.Mechanism of Fine-tuning pH Sensors in Proprotein Convertases: IDENTIFICATION OF A pH-SENSING HISTIDINE PAIR IN THE PROPEPTIDE OF PROPROTEIN CONVERTASE 1/3.The intracellular Ca²⁺ channels of membrane traffic.Optogenetic acidification of synaptic vesicles and lysosomes.TRPMLs: in sickness and in health.Revisiting the role of cystic fibrosis transmembrane conductance regulator and counterion permeability in the pH regulation of endocytic organelles.Single-cell intracellular nano-pH probes.Disease-causing mutations in the cystic fibrosis transmembrane conductance regulator determine the functional responses of alveolar macrophagesMutations in the Drosophila ortholog of the vertebrate Golgi pH regulator (GPHR) protein disturb endoplasmic reticulum and Golgi organization and affect systemic growth.Golgi pH, its regulation and roles in human disease.Proteoglycan synthesis and Golgi organization in polarized epithelial cells.Hepatocyte growth factor secreted by bone marrow stem cell reduce ER stress and improves repair in alveolar epithelial II cells.Pharmacological modulation of cGMP levels by phosphodiesterase 5 inhibitors as a therapeutic strategy for treatment of respiratory pathology in cystic fibrosis.Genetic and environmental factors influencing glutathione homeostasis in Saccharomyces cerevisiae.Silencing bcl-2 expression in epithelial cancer cells using "smart" particlesCathepsin E deficiency induces a novel form of lysosomal storage disorder showing the accumulation of lysosomal membrane sialoglycoproteins and the elevation of lysosomal pH in macrophages.A weak base-generating system suitable for selective manipulation of lysosomal pH.A smart drug: a pH-responsive photothermal ablation agent for Golgi apparatus activated cancer therapy.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Organelle acidification and disease.
@ast
Organelle acidification and disease.
@en
type
label
Organelle acidification and disease.
@ast
Organelle acidification and disease.
@en
prefLabel
Organelle acidification and disease.
@ast
Organelle acidification and disease.
@en
P2860
P1433
P1476
Organelle acidification and disease.
@en
P2093
Ora A Weisz
P2860
P356
10.1034/J.1600-0854.2003.40201.X
P577
2003-02-01T00:00:00Z