Activation-specific metabolic requirements for NK Cell IFN-γ production.
about
Role of metabolism during viral infections, and crosstalk with the innate immune systemDynamic Metabolism in Immune ResponseIL-15 activates mTOR and primes stress-activated gene expression leading to prolonged antitumor capacity of NK cells.Metabolic Regulation of Natural Killer Cell IFN-γ Production.What Fuels Natural Killers? Metabolism and NK Cell ResponsesMonocyte/Macrophage: NK Cell Cooperation-Old Tools for New Functions.Metabolism in Immune Cell Differentiation and Function.Short Term Hypoxia Synergizes with Interleukin 15 Priming in Driving Glycolytic Gene Transcription and Supports Human Natural Killer Cell ActivitiesThe Four-Way Stop Sign: Viruses, 12-Lipoxygenase, Islets, and Natural Killer Cells in Type 1 Diabetes ProgressionHigh mTOR activity is a hallmark of reactive natural killer cells and amplifies early signaling through activating receptors.Bioenergetic analysis of human peripheral blood mononuclear cells.Regulation of mTOR, Metabolic Fitness, and Effector Functions by Cytokines in Natural Killer Cells.Glycolytic requirement for NK cell cytotoxicity and cytomegalovirus control.Metabolic Adaptation of Human CD4+ and CD8+ T-Cells to T-Cell Receptor-Mediated Stimulation.Metabolic Regulation of Innate Lymphoid Cell-Mediated Tissue Protection-Linking the Nutritional State to Barrier Immunity.Continuous treatment with IL-15 exhausts human NK cells via a metabolic defect.Immune Cell Metabolism in Tumor Microenvironment.Innate Lymphoid Cell Immunometabolism.Assessment of NK Cell Metabolism.Interferon-Gamma at the Crossroads of Tumor Immune Surveillance or Evasion.Differential Reliance on Lipid Metabolism as a Salvage Pathway Underlies Functional Differences of T Cell Subsets in Poor Nutrient Environments.Expression of Interferon γ by Decidual Cells and Natural Killer Cells at the Human Implantation Site: Implications for Preeclampsia, Spontaneous Abortion, and Intrauterine Growth Restriction.IQ Domain-Containing GTPase-Activating Protein 1 Regulates Cytoskeletal Reorganization and Facilitates NKG2D-Mediated Mechanistic Target of Rapamycin Complex 1 Activation and Cytokine Gene Translation in Natural Killer Cells.Hypoxia, Metabolism and Immune Cell Function.Modulating Iron for Metabolic Support of TB Host DefenseAdenosinergic Signaling Alters Natural Killer Cell Functional ResponsesCrosstalks between mTORC1 and mTORC2 variagate cytokine signaling to control NK maturation and effector function
P2860
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P2860
Activation-specific metabolic requirements for NK Cell IFN-γ production.
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@ast
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@en
type
label
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@ast
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@en
prefLabel
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@ast
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@en
P2093
P2860
P356
P1476
Activation-specific metabolic requirements for NK Cell IFN-γ production.
@en
P2093
Annelise Y Mah
Megan A Cooper
Molly P Keppel
Nermina Saucier
Tiphanie P Vogel
P2860
P304
P356
10.4049/JIMMUNOL.1402099
P407
P577
2015-01-16T00:00:00Z