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Programmed necrosis in acute kidney injurySoluble Fn14 Is Detected and Elevated in Mouse and Human Kidney DiseaseA Bioinformatics Resource for TWEAK-Fn14 Signaling PathwayPGC1α plays a critical role in TWEAK-induced cardiac dysfunction.TWEAK/Fn14 Activation Participates in Skin Inflammation.Tnfrsf12a-Mediated Atherosclerosis Signaling and Inflammatory Response as a Common Protection Mechanism of Shuxuening Injection Against Both Myocardial and Cerebral Ischemia-Reperfusion Injuries.
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description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
TWEAK-Fn14 as a mediator of acute kidney injury.
@ast
TWEAK-Fn14 as a mediator of acute kidney injury.
@en
type
label
TWEAK-Fn14 as a mediator of acute kidney injury.
@ast
TWEAK-Fn14 as a mediator of acute kidney injury.
@en
prefLabel
TWEAK-Fn14 as a mediator of acute kidney injury.
@ast
TWEAK-Fn14 as a mediator of acute kidney injury.
@en
P2860
P356
P1433
P1476
TWEAK-Fn14 as a mediator of acute kidney injury.
@en
P2093
Joel M Weinberg
P2860
P2888
P304
P356
10.1038/KI.2010.435
P407
P577
2011-01-01T00:00:00Z
P5875
P6179
1023788999