Sequential dosing in chemosensitization: targeting the PI3K/Akt/mTOR pathway in neuroblastoma.
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Cell Proliferation in NeuroblastomaA critical evaluation of PI3K inhibition in Glioblastoma and Neuroblastoma therapyKilling me softly--future challenges in apoptosis researchA Potential Role for the Inhibition of PI3K Signaling in Glioblastoma Therapy.Role of autophagy in the ω-3 long chain polyunsaturated fatty acid-induced death of lung cancer A549 cells.Targeted Inhibition of Phosphoinositide 3-Kinase/Mammalian Target of Rapamycin Sensitizes Pancreatic Cancer Cells to Doxorubicin without Exacerbating Cardiac ToxicitySequential Therapy with Minocycline and Candesartan Improves Long-Term Recovery After Experimental Stroke.A signaling visualization toolkit to support rational design of combination therapies and biomarker discovery: SiViT.Combination of lentivirus-mediated silencing of PPM1D and temozolomide chemotherapy eradicates malignant glioma through cell apoptosis and cell cycle arrest.Neuroblastoma treatment in the post-genomic era.PI3K/Akt signaling pathway triggers P2X7 receptor expression as a pro-survival factor of neuroblastoma cells under limiting growth conditions.Thymosin alpha 1 suppresses proliferation and induces apoptosis in breast cancer cells through PTEN-mediated inhibition of PI3K/Akt/mTOR signaling pathway.The P2X7 receptor is a key modulator of the PI3K/GSK3β/VEGF signaling network: evidence in experimental neuroblastoma.New methods to control neuroblastoma growth.Delayed Sequential Co-Delivery of Gefitinib and Doxorubicin for Targeted Combination Chemotherapy.GSK3β-dependent cyclin D1 and cyclin E1 degradation is indispensable for NVP-BEZ235 induced G0/G1 arrest in neuroblastoma cells.Cell death-based treatment of neuroblastoma.The effects of PI3K-mediated signalling on glioblastoma cell behaviour.Silencing Intersectin 1 Slows Orthotopic Neuroblastoma Growth in Mice.miR‑21‑5p confers doxorubicin resistance in gastric cancer cells by targeting PTEN and TIMP3.p53 Nongenotoxic Activation and mTORC1 Inhibition Lead to Effective Combination for Neuroblastoma Therapy.Cell death-based treatment of childhood cancer.Inhibition of PI3K signalling increases the efficiency of radiotherapy in glioblastoma cellsPrecision medicine in pediatric oncology
P2860
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P2860
Sequential dosing in chemosensitization: targeting the PI3K/Akt/mTOR pathway in neuroblastoma.
description
2013 nî lūn-bûn
@nan
2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@ast
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@en
type
label
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@ast
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@en
prefLabel
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@ast
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@en
P2093
P2860
P50
P1433
P1476
Sequential dosing in chemosens ...... mTOR pathway in neuroblastoma.
@en
P2093
Claudia Jennewein
Daniela Marx
Lisa Nonnenmacher
Najmeh Faham
Patrick Gonzalez
P2860
P304
P356
10.1371/JOURNAL.PONE.0083128
P407
P577
2013-12-31T00:00:00Z