Origin of interstitial fibroblasts in an accelerated model of angiotensin II-induced renal fibrosis. - Wikidata - wikimedia - TriplyDB

Origin of interstitial fibroblasts in an accelerated model of angiotensin II-induced renal fibrosis.

Origin of interstitial fibroblasts in an accelerated model of angiotensin II-induced renal fibrosis.

http://www.wikidata.org/entity/Q35085426

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Epithelial-to-Mesenchymal Transition in Diabetic Nephropathy: Fact or Fiction?Ribosomal biogenesis induction by high glucose requires activation of upstream binding factor in kidney glomerular epithelial cells Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis NAD(P)H oxidase mediates TGF-beta1-induced activation of kidney myofibroblasts.Autophagy is a component of epithelial cell fate in obstructive uropathy.Mechanisms of disease: epithelial-mesenchymal transition--does cellular plasticity fuel neoplastic progression?RhoA/Rho kinase mediates TGF-β1-induced kidney myofibroblast activation through Poldip2/Nox4-derived reactive oxygen species.Tubular overexpression of transforming growth factor-beta1 induces autophagy and fibrosis but not mesenchymal transition of renal epithelial cells Ultrastructural study of electron dense deposits in renal tubular basement membrane: prevalence and relationship to epithelial atrophy.Acute hyperglycemia rapidly stimulates VEGF mRNA translation in the kidney. Role of angiotensin type 2 receptor (AT2).Kidney pericytes: roles in regeneration and fibrosis.AMP-activated protein kinase (AMPK) negatively regulates Nox4-dependent activation of p53 and epithelial cell apoptosis in diabetes.Pericytes at the intersection between tissue regeneration and pathology.Type of MRI contrast, tissue gadolinium, and fibrosis.Epithelial to mesenchymal transition as a biomarker in renal fibrosis: are we ready for the bedside?The role played by perivascular cells in kidney interstitial injury Reciprocal induction of simple organogenesis by mouse kidney progenitor cells in three-dimensional co-culture.Regulation of elongation phase of mRNA translation in diabetic nephropathy: amelioration by rapamycin Cholangiocytes with mesenchymal features contribute to progressive hepatic fibrosis of the polycystic kidney rat Relevance of the stroma and epithelial-mesenchymal transition (EMT) for the rheumatic diseases.Transforming growth factor β-1 stimulates profibrotic epithelial signaling to activate pericyte-myofibroblast transition in obstructive kidney fibrosis.Mechanisms of disease: Fibroblasts--a new look at an old problem.Mammalian target of rapamycin regulates Nox4-mediated podocyte depletion in diabetic renal injury.Stem cells in airway smooth muscle: state of the art.Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.Renal fibrosis is attenuated by targeted disruption of KCa3.1 potassium channels.mTORC2 Signaling Regulates Nox4-Induced Podocyte Depletion in Diabetes.Microvascular pericytes in healthy and diseased kidneys.Pathogenesis of tubulointerstitial fibrosis in chronic allograft dysfunction.Myofibroblast differentiation during fibrosis: role of NAD(P)H oxidases.The origin of interstitial myofibroblasts in chronic kidney disease.The role of cell plasticity in progression and reversal of renal fibrosis.Fibroblasts and myofibroblasts in renal fibrosis.Epithelial-mesenchymal transition (EMT) in kidney fibrosis: fact or fantasy?Origins of fibrosis: pericytes take centre stage.Epithelial-mesenchymal transition in lung development and disease: does it exist and is it important?The complexity of cell composition of the intima of large arteries: focus on pericyte-like cells.Polymorphisms in the 3' UTR in the neurocalcin delta gene affect mRNA stability, and confer susceptibility to diabetic nephropathy.Diverse origins of the myofibroblast—implications for kidney fibrosis.Tubular atrophy in the pathogenesis of chronic kidney disease progression.

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P2860

Origin of interstitial fibroblasts in an accelerated model of angiotensin II-induced renal fibrosis.

http://www.wikidata.org/entity/Q35085426

description

2005 nî lūn-bûn

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2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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The American Journal of Pathology

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Origin of interstitial fibrobl ...... sin II-induced renal fibrosis.

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P2093

Fredyne Springer

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Jeffrey L Barnes

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Jennifer L Faulkner

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Lisa M Szcykalski

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P2860

Epithelial-mesenchymal transition and its implications for fibrosis

RANTES and monocyte chemoattractant protein-1 (MCP-1) play an important role in the inflammatory phase of crescentic nephritis, but only MCP-1 is involved in crescent formation and interstitial fibrosis

Systemic infusion of angiotensin II into normal rats activates nuclear factor-kappaB and AP-1 in the kidney: role of AT(1) and AT(2) receptors

Evidence that fibroblasts derive from epithelium during tissue fibrosis

Angiotensin II-induced mitogenesis of spontaneously hypertensive rat-derived cultured smooth muscle cells is dependent on autocrine production of transforming growth factor-beta.

Bone marrow is a reservoir of repopulating mesangial cells during glomerular remodeling.

The role of angiotensin II and plasminogen activator inhibitor-1 in progressive glomerulosclerosis.

Renoprotective benefits of RAS inhibition: from ACEI to angiotensin II antagonists.

The role of vasoactive compounds, growth factors and cytokines in the progression of renal disease.

Early role of Fsp1 in epithelial-mesenchymal transformation.

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1193-1205

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