Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy. - Wikidata - wikimedia - TriplyDB

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

http://www.wikidata.org/entity/Q35093740

about

How to form and close the brain: insight into the mechanism of cranial neural tube closure in mammals Does Notch play a tumor suppressor role across diverse squamous cell carcinomas?The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?Synergistic Effect and Molecular Mechanisms of Traditional Chinese Medicine on Regulating Tumor Microenvironment and Cancer Cells Mesenchymal Stem/Stromal Cells in Stromal Evolution and Cancer Progression The function, regulation and therapeutic implications of the tumor suppressor protein, PML Non-autonomous overgrowth by oncogenic niche cells: Cellular cooperation and competition in tumorigenesis Spreading the word: non-autonomous effects of apoptosis during development, regeneration and disease Imaging radiation response in tumor and normal tissue.The impact of the myeloid response to radiation therapy Biomarkers and molecular probes for cell death imaging and targeted therapeutics How cell death shapes cancer Killing me softly--future challenges in apoptosis research Radiation as immunomodulator: implications for dendritic cell-based immunotherapy Caspase-3 promotes genetic instability and carcinogenesis.Old, new and emerging functions of caspases.Peripheral blood mononuclear cell secretome for tissue repair Cyclooxygenase-2: A Role in Cancer Stem Cell Survival and Repopulation of Cancer Cells during Therapy Metastatic colonization by circulating tumour cells The paradox role of caspase cascade in ionizing radiation therapy Bioactive lipids and the control of Bax pro-apoptotic activity Systems biology of cancer: entropy, disorder, and selection-driven evolution to independence, invasion and "swarm intelligence"Inhibitory effects and underlying mechanism of 7-hydroxyflavone phosphate ester in HeLa cells Cell survival signalling through PPARδ and arachidonic acid metabolites in neuroblastoma Key roles of necroptotic factors in promoting tumor growth Apoptotic DNA degradation into oligonucleosomal fragments, but not apoptotic nuclear morphology, relies on a cytosolic pool of DFF40/CAD endonuclease CDH1 and IL1-beta expression dictates FAK and MAPKK-dependent cross-talk between cancer cells and human mesenchymal stem cells.Meta-dimensional data integration identifies critical pathways for susceptibility, tumorigenesis and progression of endometrial cancer Inosine Released from Dying or Dead Cells Stimulates Cell Proliferation via Adenosine Receptors.Caspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.Differential sensitivity to apoptosome apparatus activation in non-small cell lung carcinoma and the lung The Drosophila effector caspase Dcp-1 regulates mitochondrial dynamics and autophagic flux via SesB The Antigastric Cancer Activity of San Leng Powder Extract Induces Apoptosis in Balb/C Bearing-SGC-7901 Mice.From computational modelling of the intrinsic apoptosis pathway to a systems-based analysis of chemotherapy resistance: achievements, perspectives and challenges in systems medicine.Three-dimensional spheroid culture targeting versatile tissue bioassays using a PDMS-based hanging drop array.Expression levels of cleaved caspase-3 and caspase-3 in tumorigenesis and prognosis of oral tongue squamous cell carcinoma.Simvastatin protects hepatocytes from apoptosis by suppressing the TNF-α/caspase-3 signaling pathway in mice with burn injury A polymorphism in JMJD2C alters the cleavage by caspase-3 and the prognosis of human breast cancer.Elevated cleaved caspase-3 is associated with shortened overall survival in several cancer types.Effect of celecoxib on inhibiting tumor repopulation during radiotherapy in human FaDu squamous cell carcinoma

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P2860

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

http://www.wikidata.org/entity/Q35093740

description

2011 nî lūn-bûn

@nan

2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

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2011年論文

@zh-mo

2011年論文

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2011年论文

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name

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

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Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

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type

label

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

@ast

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

@en

prefLabel

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

@ast

Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

@en

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Nature Medicine

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Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.

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Aik-Choon Tan

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Andrew Thorburn

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Antonio Jimeno

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Chuan-Yuan Li

http://www.w3.org/2001/XMLSchema#string

David Raben

http://www.w3.org/2001/XMLSchema#string

Fang Li

http://www.w3.org/2001/XMLSchema#string

Gangwen Han

http://www.w3.org/2001/XMLSchema#string

Jackie Thorburn

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Jingping Shen

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Joel S Bedford

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P2860

Biochemical characteristics of caspases-3, -6, -7, and -8

Caspase-3-dependent activation of calcium-independent phospholipase A2 enhances cell migration in non-apoptotic ovarian cancer cells

Caspase-8 serves both apoptotic and nonapoptotic roles

Caspases 3 and 7: key mediators of mitochondrial events of apoptosis

Apoptosis: controlled demolition at the cellular level

Use of reporter genes for optical measurements of neoplastic disease in vivo

Apoptotic cells activate the "phoenix rising" pathway to promote wound healing and tissue regeneration

Regulation of HIF-1alpha stability through S-nitrosylation.

Apoptotic cells induce migration of phagocytes via caspase-3-mediated release of a lipid attraction signal.

Matrix metalloproteinase-9 is required for tumor vasculogenesis but not for angiogenesis: role of bone marrow-derived myelomonocytic cells

P2888

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860-866

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scholarly article

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10.1038/NM.2385

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P433

7

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17

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Brian O'Sullivan

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2011-07-03T00:00:00Z

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51463553

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21725296

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radiation therapy

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3132290

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