Differential clearance and host-pathogen interactions of YopE- and YopK- YopL- Yersinia pestis in BALB/c mice.
about
The V antigen of Yersinia pestis regulates Yop vectorial targeting as well as Yop secretion through effects on YopB and LcrG.Type III protein secretion systems in bacterial pathogens of animals and plantsDifferential effects of deletions in lcrV on secretion of V antigen, regulation of the low-Ca2+ response, and virulence of Yersinia pestisThe virulence plasmid of Yersinia, an antihost genomeYersinia pestis--etiologic agent of plagueYersinia pestis YopM: thrombin binding and overexpression.Yersinia has a tropism for B and T cell zones of lymph nodes that is independent of the type III secretion system.Absence of inflammation and pneumonia during infection with nonpigmented Yersinia pestis reveals a new role for the pgm locus in pathogenesis.Immune defense against pneumonic plagueThe Yersinia pestis YscY protein directly binds YscX, a secreted component of the type III secretion machinery.Phosphoglucomutase of Yersinia pestis is required for autoaggregation and polymyxin B resistance.YopR impacts type III needle polymerization in Yersinia species.The C-terminal tail of Yersinia pseudotuberculosis YopM is critical for interacting with RSK1 and for virulenceROS-inhibitory activity of YopE is required for full virulence of Yersinia in miceProtective efficacy of recombinant Yersinia outer proteins against bubonic plague caused by encapsulated and nonencapsulated Yersinia pestisAnti-LcrV antibody inhibits delivery of Yops by Yersinia pestis KIM5 by directly promoting phagocytosis.Genome-wide mutant fitness profiling identifies nutritional requirements for optimal growth of Yersinia pestis in deep tissue.Three Yersinia pestis adhesins facilitate Yop delivery to eukaryotic cells and contribute to plague virulenceThe proinflammatory response induced by wild-type Yersinia pseudotuberculosis infection inhibits survival of yop mutants in the gastrointestinal tract and Peyer's patches.Association between virulence of Yersinia pestis and suppression of gamma interferon and tumor necrosis factor alpha.Passive immunity to yersiniae mediated by anti-recombinant V antigen and protein A-V antigen fusion peptidePhysiological basis of the low calcium response in Yersinia pestis.Early apoptosis of macrophages modulated by injection of Yersinia pestis YopK promotes progression of primary pneumonic plagueIL-17 contributes to cell-mediated defense against pulmonary Yersinia pestis infection.Contributions of chaperone/usher systems to cell binding, biofilm formation and Yersinia pestis virulence.Yersinia pestis YopJ suppresses tumor necrosis factor alpha induction and contributes to apoptosis of immune cells in the lymph node but is not required for virulence in a rat model of bubonic plagueYopM inhibits platelet aggregation and is necessary for virulence of Yersinia pestis in mice.Requirement of the Yersinia pseudotuberculosis effectors YopH and YopE in colonization and persistence in intestinal and lymph tissuesVirulence plasmid-encoded YopK is essential for Yersinia pseudotuberculosis to cause systemic infection in mice.Active immunization with recombinant V antigen from Yersinia pestis protects mice against plagueSuppression of cytokines in mice by protein A-V antigen fusion peptide and restoration of synthesis by active immunization.Invasin production by Yersinia pestis is abolished by insertion of an IS200-like element within the inv gene.Resistance to lipopolysaccharide mediated by the Yersinia pestis V antigen-polyhistidine fusion peptide: amplification of interleukin-10.A protective epitope in type III effector YopE is a major CD8 T cell antigen during primary infection with Yersinia pseudotuberculosis.Intranasal inoculation of mice with Yersinia pseudotuberculosis causes a lethal lung infection that is dependent on Yersinia outer proteins and PhoP.A novel post-exposure medical countermeasure L-97-1 improves survival and acute lung injury following intratracheal infection with Yersinia pestisStructure and regulation of the Yersinia pestis yscBCDEF operon.The Yersinia pestis V antigen is a regulatory protein necessary for Ca2(+)-dependent growth and maximal expression of low-Ca2+ response virulence genesLcrD, a membrane-bound regulator of the Yersinia pestis low-calcium responseThe plague virulence protein YopM targets the innate immune response by causing a global depletion of NK cells
P2860
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P2860
Differential clearance and host-pathogen interactions of YopE- and YopK- YopL- Yersinia pestis in BALB/c mice.
description
1989 nî lūn-bûn
@nan
1989 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@ast
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@en
type
label
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@ast
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@en
prefLabel
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@ast
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@en
P2860
P1476
Differential clearance and hos ...... ersinia pestis in BALB/c mice.
@en
P2093
M L Cibull
S C Straley
P2860
P304
P407
P577
1989-04-01T00:00:00Z