Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis. - Wikidata - wikimedia - TriplyDB

Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis.

Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis.

http://www.wikidata.org/entity/Q35094492

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G protein-coupled receptor 30-dependent protein kinase A pathway is critical in nongenomic effects of estrogen in attenuating liver injury after trauma-hemorrhage Is boosting the immune system in sepsis appropriate?Delivery of RNAi Therapeutics to the Airways-From Bench to Bedside Aerosol Delivery of siRNA to the Lungs. Part 1: Rationale for Gene Delivery Systems Protection against sepsis-induced lung injury by selective inhibition of protein kinase C-δ (δ-PKC)Fas inhibition attenuates lipopolysaccharide-induced apoptosis and cytokine release of rat type II alveolar epithelial cells A comparison of vasopressin, terlipressin, and lactated ringers for resuscitation of uncontrolled hemorrhagic shock in an animal model.Plasmacytoid dendritic cells control lung inflammation and monocyte recruitment in indirect acute lung injury in mice Differential role of the Fas/Fas ligand apoptotic pathway in inflammation and lung fibrosis associated with reovirus 1/L-induced bronchiolitis obliterans organizing pneumonia and acute respiratory distress syndrome.Pulmonary gene silencing in transgenic EGFP mice using aerosolised chitosan/siRNA nanoparticles.LPS-induced systemic inflammation is more severe in P2Y12 null mice.Ciglitazone, a novel inhibitor of lung apoptosis following hemorrhagic shock.Febrile-range hyperthermia augments lipopolysaccharide-induced lung injury by a mechanism of enhanced alveolar epithelial apoptosis Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival.Comparison of the effects of aging and IL-6 on the hepatic inflammatory response in two models of systemic injury: scald injury versus i.p. LPS administration Cathepsin B is activated as an executive protease in fetal rat alveolar type II cells exposed to hyperoxia.The immune response after fracture trauma is different in old compared to young patients.Pathogenesis of indirect (secondary) acute lung injury.Experimental models and emerging hypotheses for acute lung injury.Fas activation in alveolar epithelial cells induces KC (CXCL1) release by a MyD88-dependent mechanism.Disruption of fas-fas ligand signaling, apoptosis, and innate immunity by bacterial pathogens Role of the Fas/FasL system in a model of RSV infection in mechanically ventilated mice.The caspase inhibitor zVAD increases lung inflammation in pneumovirus infection in mice Clinical review: gene-based therapies for ALI/ARDS: where are we now?Mechanisms of indirect acute lung injury: a novel role for the coinhibitory receptor, programmed death-1.The contribution of CD4+ CD25+ T-regulatory-cells to immune suppression in sepsis Epithelial apoptosis in mechanistically distinct methods of injury in the murine small intestine The role and source of tumor necrosis factor-α in hemorrhage-induced priming for septic lung injury.Deletion of apoptosis signal-regulating kinase-1 prevents ventilator-induced lung injury in mice.Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.Role of Complement C5 in Experimental Blunt Chest Trauma-Induced Septic Acute Lung Injury (ALI).Mechanisms of methicillin-resistant Staphylococcus aureus pneumonia-induced intestinal epithelial apoptosis Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury Delivery systems for the direct application of siRNAs to induce RNA interference (RNAi) in vivo.Fas-ligand-induced apoptosis of respiratory epithelial cells causes disruption of postcanalicular alveolar development Blockade of apoptosis as a rational therapeutic strategy for the treatment of sepsis Immunological research using RNA interference technology.Applications of RNA interference: current state and prospects for siRNA-based strategies in vivo.Interfering with disease: a progress report on siRNA-based therapeutics.Depletion of resident alveolar macrophages does not prevent Fas-mediated lung injury in mice.

P2860

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P2860

Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis.

http://www.wikidata.org/entity/Q35094492

description

2005 nî lūn-bûn

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2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

@zh-hk

2005年論文

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2005年論文

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2005年论文

@wuu

name

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@ast

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@en

type

label

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@ast

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@en

prefLabel

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@ast

Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@en

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The American Journal of Pathology

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Silencing of Fas, but not casp ...... hemorrhagic shock and sepsis.

@en

P2093

Chun-Shiang Chung

http://www.w3.org/2001/XMLSchema#string

Joanne Lomas-Neira

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Mario Perl

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Tina-Marie Rachel

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Walter L Biffl

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William G Cioffi

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P2860

Sequence-specific potent induction of IFN-alpha by short interfering RNA in plasmacytoid dendritic cells through TLR7

Caspase 8 small interfering RNA prevents acute liver failure in mice

Fas and fas ligand are up-regulated in pulmonary edema fluid and lung tissue of patients with acute lung injury and the acute respiratory distress syndrome

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

Fas ligation on macrophages enhances IL-1R1-Toll-like receptor 4 signaling and promotes chronic inflammation

Activation of the interferon system by short-interfering RNAs

Small interfering RNA (siRNA) targeting VEGF effectively inhibits ocular neovascularization in a mouse model.

Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria

Regulation of lymphocyte proliferation and death by FLIP.

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