Leukocyte-derived interleukin-1beta interacts with renal interleukin-1 receptor I to promote renal tumor necrosis factor and glomerular injury in murine crescentic glomerulonephritis - Wikidata - wikimedia - TriplyDB

Leukocyte-derived interleukin-1beta interacts with renal interleukin-1 receptor I to promote renal tumor necrosis factor and glomerular injury in murine crescentic glomerulonephritis

Leukocyte-derived interleukin-1beta interacts with renal interleukin-1 receptor I to promote renal tumor necrosis factor and glomerular injury in murine crescentic glomerulonephritis

http://www.wikidata.org/entity/Q35098385

about

Proinflammatory Cytokines and Potassium Channels in the Kidney Anti-GBM glomerulonephritis involves IL-1 but is independent of NLRP3/ASC inflammasome-mediated activation of caspase-1.Urinary elafin and kidney injury in hematopoietic cell transplant recipients Distinct contributions of TNF receptor 1 and 2 to TNF-induced glomerular inflammation in mice.Neutrophil serine proteases promote IL-1β generation and injury in necrotizing crescentic glomerulonephritis.Protective effects of genetic inhibition of Discoidin Domain Receptor 1 in experimental renal disease Predictive value of conjointly examined IL-1ra, TNF-R I, TNF-R II, and RANTES in patients with primary glomerulonephritis.Leukocytes in glomerular injury.Activated renal macrophages are markers of disease onset and disease remission in lupus nephritis.Kallikrein transduced mesenchymal stem cells protect against anti-GBM disease and lupus nephritis by ameliorating inflammation and oxidative stress Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.Activation of inflammasomes in podocyte injury of mice on the high fat diet: Effects of ASC gene deletion and silencing How anti-neutrophil cytoplasmic autoantibodies activate neutrophils.Canonical and non-canonical effects of the NLRP3 inflammasome in kidney inflammation and fibrosis.Of Inflammasomes and Alarmins: IL-1β and IL-1α in Kidney Disease.Anti-tumor necrosis factor α treatment of interferon-α-induced murine lupus nephritis reduces the renal macrophage response but does not alter glomerular immune complex formation.Overexpression of Toll-like receptor 8 correlates with the progression of podocyte injury in murine autoimmune glomerulonephritis.An altered TH1/TH2 and pro-inflammatory cytokine profile in patients with end-stage renal disease detected by suspension array technology.Interleukin-1β suppresses activity of an inwardly rectifying K+ channel in human renal proximal tubule cells.IL-1RI deficiency ameliorates early experimental renal interstitial fibrosis.Interleukin-1 cluster gene polymorphisms in childhood IgA nephropathy.Anti-C1q Autoantibodies from Systemic Lupus Erythematosus Patients Induce a Proinflammatory Phenotype in Macrophages.Dexamethasone pretreatment attenuates lung and kidney injury in cholestatic rats induced by hepatic ischemia/reperfusion.

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Leukocyte-derived interleukin-1beta interacts with renal interleukin-1 receptor I to promote renal tumor necrosis factor and glomerular injury in murine crescentic glomerulonephritis

http://www.wikidata.org/entity/Q35098385

description

2004 nî lūn-bûn

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2004 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի հունիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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The American Journal of Pathology

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Leukocyte-derived interleukin- ...... crescentic glomerulonephritis

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A Richard Kitching

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Jennifer R Timoshanko

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Peter G Tipping

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Stephen R Holdsworth

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Yichiro Iwakura

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P2860

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Glomerular tumor necrosis factor and interleukin 1 during acute aminonucleoside nephrosis. An immunohistochemical study

Th1 and Th2 T helper cell subsets affect patterns of injury and outcomes in glomerulonephritis.

Perivascular microglial cells of the CNS are bone marrow-derived and present antigen in vivo.

Tumor necrosis factor-alpha is expressed by glomerular visceral epithelial cells in human membranous nephropathy.

Interleukin-1 beta and tumor necrosis factor-alpha induce gene expression and production of leukocyte chemotactic factors, colony-stimulating factors, and interleukin-6 in human mesangial cells.

Tumor necrosis factor-alpha mRNA expression in lipopolysaccharide-stimulated rat kidney. Chronological analysis of localization

Interleukin-1 beta gene expression in experimental glomerulonephritis in the rat: an in-situ hybridization study.

The clinical course of experimental autoimmune encephalomyelitis and inflammation is controlled by the expression of CD40 within the central nervous system.

Production of mice deficient in genes for interleukin (IL)-1alpha, IL-1beta, IL-1alpha/beta, and IL-1 receptor antagonist shows that IL-1beta is crucial in turpentine-induced fever development and glucocorticoid secretion.

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1967-1977

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2004-06-01T00:00:00Z

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15161633

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glomerulonephritis

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1615771

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