Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors. - Wikidata - wikimedia - TriplyDB

Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

http://www.wikidata.org/entity/Q35102867

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MicroRNAs in Nonalcoholic Fatty Liver Disease Key discoveries in bile acid chemistry and biology and their clinical applications: history of the last eight decades The G-protein coupled bile salt receptor TGR5 is expressed in liver sinusoidal endothelial cells Activation of focal adhesion kinase and JNK contributes to the extracellular matrix and cAMP-GEF mediated survival from bile acid induced apoptosis in rat hepatocytes Overexpression of mcl-1 attenuates liver injury and fibrosis in the bile duct-ligated mouse.Hepatocyte death: a clear and present danger.Tumor necrosis factor-α promotes cholestasis-induced liver fibrosis in the mouse through tissue inhibitor of metalloproteinase-1 production in hepatic stellate cells.Death receptor 5 signaling promotes hepatocyte lipoapoptosis Na(+) /H(+) exchanger regulatory factor 1 knockout mice have an attenuated hepatic inflammatory response and are protected from cholestatic liver injury Individuals with Primary Sclerosing Cholangitis Have Elevated Levels of Biomarkers for Apoptosis but Not Necrosis.Inhibition of NF-κB by deoxycholic acid induces miR-21/PDCD4-dependent hepatocelular apoptosis.A metabolomic perspective of griseofulvin-induced liver injury in mice.Prediction of drug-induced intrahepatic cholestasis: in vitro screening and QSAR analysis of drugs inhibiting the human bile salt export pump.Detachment of esophageal carcinoma cells from extracellular matrix causes relocalization of death receptor 5 and apoptosis.FXR: a metabolic regulator and cell protector.Hepatic 3D spheroid models for the detection and study of compounds with cholestatic liability.Technical pitfalls and improvements for high-speed screening and QSAR analysis to predict inhibitors of the human bile salt export pump (ABCB11/BSEP).Plasma biomarkers of liver injury and inflammation demonstrate a lack of apoptosis during obstructive cholestasis in mice Mechanisms of liver disease: cross-talk between the NF-kappaB and JNK pathways.Osteopontin is an initial mediator of inflammation and liver injury during obstructive cholestasis after bile duct ligation in mice.c-Jun N-terminal kinase 1/c-Jun activation of the p53/microRNA 34a/sirtuin 1 pathway contributes to apoptosis induced by deoxycholic acid in rat liver.Chenodeoxycholic acid activates NLRP3 inflammasome and contributes to cholestatic liver fibrosis.Apoptosis and modulation of cell cycle control by bile acids in human leukemia T cells.Characterization of enantiomeric bile acid-induced apoptosis in colon cancer cell lines.Overexpression of the cholesterol-binding protein MLN64 induces liver damage in the mouse.Eupatilin attenuates bile acid-induced hepatocyte apoptosis.The future of GI and liver research: editorial perspectives. III. JNK/AP-1 regulation of hepatocyte death.Cloning of the dog bile salt export pump (BSEP; ABCB11) and functional comparison with the human and rat proteins.Metformin protects rat hepatocytes against bile acid-induced apoptosis Protein kinase C induces endocytosis of the sodium taurocholate cotransporting polypeptide.Role of acid sphingomyelinase of Kupffer cells in cholestatic liver injury in mice.Selected contribution: Effects of aging on cerebrovascular tone and [Ca2+]i.What doesn't kill you makes you stronger: how hepatocytes survive prolonged cholestasis.Growth-dependent changes in endothelial factors regulating arteriolar tone.Increased myogenic responsiveness of skeletal muscle arterioles with juvenile growth.Ionic mechanisms mediating the myogenic response in newborn porcine cerebral arteries.Maturational modulation of endothelium-dependent vasodilatation in ovine cerebral arteries.Effects of carbon monoxide and heme oxygenase inhibitors in cerebral vessels of rats and mice.Activation of cAMP-guanine exchange factor confers PKA-independent protection from hepatocyte apoptosis.Living-related liver transplantation for siblings with progressive familial intrahepatic cholestasis 2, with novel genetic findings.

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

http://www.wikidata.org/entity/Q35102867

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2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2003 թվականի մայիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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type

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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AJPGI.00491.2002

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors.

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Hajime Higuchi

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P2860

Caspase-10 is recruited to and activated at the native TRAIL and CD95 death-inducing signalling complexes in a FADD-dependent manner but can not functionally substitute caspase-8.

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

Two CD95 (APO-1/Fas) signaling pathways

Bile acid-induced rat hepatocyte apoptosis is inhibited by antioxidants and blockers of the mitochondrial permeability transition.

Ultraviolet light induces apoptosis via direct activation of CD95 (Fas/APO-1) independently of its ligand CD95L

Toxic bile salts induce rodent hepatocyte apoptosis via direct activation of Fas.

Deoxycholic acid (DCA) causes ligand-independent activation of epidermal growth factor receptor (EGFR) and FAS receptor in primary hepatocytes: inhibition of EGFR/mitogen-activated protein kinase-signaling module enhances DCA-induced apoptosis.

Is TRAIL hepatotoxic?

Herpes simplex virus thymidine kinase/ganciclovir-induced apoptosis involves ligand-independent death receptor aggregation and activation of caspases.

Fas enhances fibrogenesis in the bile duct ligated mouse: a link between apoptosis and fibrosis.

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10.1152/AJPGI.00491.2002

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Gregory J Gores

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