Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
about
The Kallikrein-Kinin System: A Novel Mediator of IL-17-Driven Anti-Candida Immunity in the KidneyNeural crest deletion of Dlx3 leads to major dentin defects through down-regulation of DsppA GRHL3-regulated repair pathway suppresses immune-mediated epidermal hyperplasiaIncreased retinoic acid levels through ablation of Cyp26b1 determine the processes of embryonic skin barrier formation and peridermal developmentDLX3 interacts with GCM1 and inhibits its transactivation-stimulating activity in a homeodomain-dependent manner in human trophoblast-derived cells.DLX3 regulates bone mass by targeting genes supporting osteoblast differentiation and mineral homeostasis in vivoIL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.Epidermal stem cells in the isthmus/infundibulum influence hair shaft differentiation: evidence from targeted DLX3 deletion.Bone marrow-derived mesenchymal stromal cells harness purinergenic signaling to tolerize human Th1 cells in vivo.Inflammation induces dermal Vγ4+ γδT17 memory-like cells that travel to distant skin and accelerate secondary IL-17-driven responses.Anticancer activity of a novel small molecule tubulin inhibitor STK899704The homeoprotein DLX3 and tumor suppressor p53 co-regulate cell cycle progression and squamous tumor growthSphingosine-1-phosphate phosphatase 1 regulates keratinocyte differentiation and epidermal homeostasisPNPLA1 has a crucial role in skin barrier function by directing acylceramide biosynthesis.A novel DLX3-PKC integrated signaling network drives keratinocyte differentiation.Tbet is a critical modulator of FoxP3 expression in autoimmune graft-versus-host diseaseDlx3 and GCM-1 functionally coordinate the regulation of placental growth factor in human trophoblast-derived cells.Transcriptional Immunoprofiling at the Tick-Virus-Host Interface during Early Stages of Tick-Borne Encephalitis Virus Transmission.Wound regeneration deficit in rats correlates with low morphogenetic potential and distinct transcriptome profile of epidermis.Homeobox transcription factor DLX4 is not necessary for skin development and homeostasis.DLX3-dependent STAT3 signaling in keratinocytes regulates skin immune homeostasis.Role of FGFR2b expression and signaling in keratinocyte differentiation: sequential involvement of PKCδ and PKCα.
P2860
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P2860
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@ast
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@en
type
label
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@ast
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@en
prefLabel
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@ast
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@en
P2093
P2860
P356
P1476
Epidermal ablation of Dlx3 is linked to IL-17-associated skin inflammation.
@en
P2093
Eung Ho Choi
Hyouk-Soo Kwon
Joonsung Hwang
Maria I Morasso
Mark C Udey
Ryosuke Kita
Seung Hun Lee
P2860
P304
11566-11571
P356
10.1073/PNAS.1019658108
P407
P577
2011-06-27T00:00:00Z