Fibrosis, not cell size, delineates beta-myosin heavy chain reexpression during cardiac hypertrophy and normal aging in vivo.
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LARP6 Meets Collagen mRNA: Specific Regulation of Type I Collagen ExpressionRNA protein interactions governing expression of the most abundant protein in human body, type I collagenCross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequencesGDF11 does not rescue aging-related pathological hypertrophyCardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxiaAdverse effects of high glucose and free fatty acid on cardiomyocytes are mediated by connective tissue growth factorActivating transcription factor 3 deficiency promotes cardiac hypertrophy, dysfunction, and fibrosis induced by pressure overloadSpeckle tracking echocardiography detects uremic cardiomyopathy early and predicts cardiovascular mortality in ESRD.Dysautonomia due to reduced cholinergic neurotransmission causes cardiac remodeling and heart failure.Aging, telomeres and heart failure.Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β.Heterogeneous myocyte enhancer factor-2 (Mef2) activation in myocytes predicts focal scarring in hypertrophic cardiomyopathy.Heart failure in elderly patients: distinctive features and unresolved issues.Androgens contribute to sex differences in myocardial remodeling under pressure overload by a mechanism involving TGF-β.Role of LARP6 and nonmuscle myosin in partitioning of collagen mRNAs to the ER membrane.Calcium dependent CAMTA1 in adult stem cell commitment to a myocardial lineageConnexin40 abnormalities and atrial fibrillation in the human heartA systematic review of fetal genes as biomarkers of cardiac hypertrophy in rodent models of diabetes.GATA6 reporter gene reveals myocardial phenotypic heterogeneity that is related to variations in gap junction coupling.The use of human cardiac tissue in biophysical research: the risks of translationAndropause and the development of cardiovascular disease presentation-more than an epi-phenomenon.Intermittent hypoxia causes NOX2-dependent remodeling of atrial connexins.Discordant on/off switching of gene expression in myocytes during cardiac hypertrophy in vivo.Nonmuscle myosin-dependent synthesis of type I collagen.Regulator of G protein signalling 14 attenuates cardiac remodelling through the MEK-ERK1/2 signalling pathway.Altered left ventricular performance in aging physically active mice with an ankle sprain injuryDecreased beta-adrenergic responsiveness following hypertrophy occurs only in cardiomyocytes that also re-express beta-myosin heavy chainCardiac fibroblast paracrine factors alter impulse conduction and ion channel expression of neonatal rat cardiomyocytesFGF2 modulates cardiac remodeling in an isoform- and sex-specific manner.MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.American Thyroid Association Guide to investigating thyroid hormone economy and action in rodent and cell models.Toll-interacting protein (Tollip) negatively regulates pressure overload-induced ventricular hypertrophy in miceInterferon regulatory factor 7 functions as a novel negative regulator of pathological cardiac hypertrophyEssential role of TEA domain transcription factors in the negative regulation of the MYH 7 gene by thyroid hormone and its receptors.Myofibroblast-mediated mechanisms of pathological remodelling of the heart.Myofibroblast secretome and its auto-/paracrine signaling.β-myosin heavy chain is induced by pressure overload in a minor subpopulation of smaller mouse cardiac myocytes.Atrophied cardiomyocytes and their potential for rescue and recovery of ventricular function.Calcific Aortic Valve Disease: Part 2-Morphomechanical Abnormalities, Gene Reexpression, and Gender Effects on Ventricular Hypertrophy and Its Reversibility.Exposure to chronic alcohol accelerates development of wall stress and eccentric remodeling in rats with volume overload.
P2860
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P2860
Fibrosis, not cell size, delineates beta-myosin heavy chain reexpression during cardiac hypertrophy and normal aging in vivo.
description
2006 nî lūn-bûn
@nan
2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@ast
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@en
type
label
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@ast
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@en
prefLabel
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@ast
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@en
P2860
P356
P1476
Fibrosis, not cell size, delin ...... ophy and normal aging in vivo.
@en
P2093
Hyung-Suk Kim
Kumar Pandya
P2860
P304
16864-16869
P356
10.1073/PNAS.0607700103
P407
P577
2006-10-26T00:00:00Z