The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity. - Wikidata - wikimedia - TriplyDB

The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

http://www.wikidata.org/entity/Q35122917

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The ToxBank Data Warehouse: Supporting the Replacement of In Vivo Repeated Dose Systemic Toxicity Testing Deoxynivalenol impairs hepatic and intestinal gene expression of selected oxidative stress, tight junction and inflammation proteins in broiler chickens, but addition of an adsorbing agent shifts the effects to the distal parts of the small intestin Quantitative Trait Loci and Candidate Genes for Neutrophil Recruitment in Sterile Inflammation Mapped in AXB-BXA Recombinant Inbred Mice Hepatic stellate cells orchestrate clearance of necrotic cells in a hypoxia-inducible factor-1α-dependent manner by modulating macrophage phenotype in mice Age-related decrease in cerebrovascular-derived neuroprotective proteins: effect of acetaminophen Effect of trifluoperazine on toxicity, HIF-1α induction and hepatocyte regeneration in acetaminophen toxicity in mice.YC-1 exerts inhibitory effects on MDA-MB-468 breast cancer cells by targeting EGFR in vitro and in vivo under normoxic condition Role of Hypoxia-Inducible Factors in the Development of Liver Fibrosis.Hypoxia and hypoxia inducible factors: diverse roles in liver diseases.An overview of transcriptional regulation in response to toxicological insult.SEURAT-1 liver gold reference compounds: a mechanism-based review.Liquid chromatography mass spectrometry-based profiling of phosphatidylcholine and phosphatidylethanolamine in the plasma and liver of acetaminophen-induced liver injured mice.Serotonin deficiency exacerbates acetaminophen-induced liver toxicity in mice.Quantitative analysis of acetaminophen and its primary metabolites in small plasma volumes by liquid chromatography-tandem mass spectrometry.Dietary α-ketoglutarate supplementation improves hepatic and intestinal energy status and anti-oxidative capacity of Cherry Valley ducks.Hepatic dysfunction and thrombocytopenia induced by excess sFlt1 in mice lacking endothelial nitric oxide synthase.

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

http://www.wikidata.org/entity/Q35122917

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2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

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Journal of Pharmacology and Experimental Therapeutics

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The role of hypoxia-inducible factor-1α in acetaminophen hepatotoxicity.

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Bryan L Copple

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Erica M Sparkenbaugh

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James P Luyendyk

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Jane F Maddox

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John J LaPres

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Krista K Greenwood

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Patricia E Ganey

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Robert A Roth

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Yogesh Saini

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P2860

Deletion of the developmentally essential gene ATR in adult mice leads to age-related phenotypes and stem cell loss

The hepatic inflammatory response after acetaminophen overdose: role of neutrophils

HIF-1 alpha is required for solid tumor formation and embryonic vascularization.

HIF-1alpha is essential for myeloid cell-mediated inflammation

c-Jun N-terminal kinase modulates oxidant stress and peroxynitrite formation independent of inducible nitric oxide synthase in acetaminophen hepatotoxicity.

Role of caspase-1 and interleukin-1beta in acetaminophen-induced hepatic inflammation and liver injury

The oxygen tension modulates acetaminophen-induced mitochondrial oxidant stress and cell injury in cultured hepatocytes

Tissue repair: an important determinant of final outcome of toxicant-induced injury.

Acetaminophen hepatotoxicity and HIF-1α induction in acetaminophen toxicity in mice occurs without hypoxia.

Hypoxia-inducible factor-dependent production of profibrotic mediators by hypoxic hepatocytes

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