Microglial Cx3cr1 knockout reduces prion disease incubation time in mice - Wikidata - wikimedia - TriplyDB

Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Pharmacological Modulation of Functional Phenotypes of Microglia in Neurodegenerative Diseases The sheddase ADAM10 is a potent modulator of prion disease.Fractalkine Receptor Deficiency Is Associated with Early Protection but Late Worsening of Outcome following Brain Trauma in Mice Knockout of fractalkine receptor Cx3cr1 does not alter disease or microglial activation in prion-infected mice Microglia in prion diseases.A roadmap for investigating the role of the prion protein in depression associated with neurodegenerative disease What Is Our Current Understanding of PrPSc-Associated Neurotoxicity and Its Molecular Underpinnings?Electron microscopic and confocal laser microscopy analysis of amyloid plaques in chronic wasting disease transmitted to transgenic mice.Absence of CCL2 and CCL3 Ameliorates Central Nervous System Grey Matter But Not White Matter Demyelination in the Presence of an Intact Blood-Brain Barrier.Immunohistochemical and Molecular Investigations Show Alteration in the Inflammatory Profile of Multiple System Atrophy Brain.

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

http://www.wikidata.org/entity/Q35127326

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2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

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1471-2202-15-44

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BMC Neuroscience

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Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

@en

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John Collinge

http://www.w3.org/2001/XMLSchema#string

Julia Grizenkova

http://www.w3.org/2001/XMLSchema#string

Sarah E Lloyd

http://www.w3.org/2001/XMLSchema#string

Sebastian Brandner

http://www.w3.org/2001/XMLSchema#string

Shaheen Akhtar

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Prion diseases of humans and animals: their causes and molecular basis

Control of microglial neurotoxicity by the fractalkine receptor

Expression of fractalkine (CX3CL1) and its receptor, CX3CR1, during acute and chronic inflammation in the rodent CNS

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

Improving bioscience research reporting: the ARRIVE guidelines for reporting animal research

Development of monocytes, macrophages, and dendritic cells

Analysis of fractalkine receptor CX(3)CR1 function by targeted deletion and green fluorescent protein reporter gene insertion

Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

Regulation of tau pathology by the microglial fractalkine receptor.

CX3CR1 deficiency alters microglial activation and reduces beta-amyloid deposition in two Alzheimer's disease mouse models.

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1471-2202-15-44

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10.1186/1471-2202-15-44

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15

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Prion protein family

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3998043

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