about
The DNA-damage response in human biology and diseaseThe Response to Oxidative DNA Damage in Neurons: Mechanisms and DiseaseMammalian transcription-coupled excision repairDisruption of aldo-keto reductase genes leads to elevated markers of oxidative stress and inositol auxotrophy in Saccharomyces cerevisiaeNucleotide excision repair in humansThe 8,5'-cyclopurine-2'-deoxynucleosides: candidate neurodegenerative DNA lesions in xeroderma pigmentosum, and unique probes of transcription and nucleotide excision repair.The evolution of transcription-associated biases of mutations across vertebratesOxidative stress triggers the preferential assembly of base excision repair complexes on open chromatin regionsCis-acting genetic variation at an E2F1/YY1 response site and putative p53 site is associated with altered allele-specific expression of ERCC5 (XPG) transcript in normal human bronchial epithelium.Novel irreversible small molecule inhibitors of replication protein A display single-agent activity and synergize with cisplatinInitiation of DNA interstrand cross-link repair in mammalian cells.Polymorphisms in the ERCC5 gene and risk of esophageal squamous cell carcinoma (ESCC) in Eastern Chinese populations.Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic diseaseTranscription restores DNA repair to heterochromatin, determining regional mutation rates in cancer genomes.DNA repair mechanisms in dividing and non-dividing cells.Terminally differentiated muscle cells are defective in base excision DNA repair and hypersensitive to oxygen injury.Photobiological Origins of the Field of Genomic Maintenance.Do all of the neurologic diseases in patients with DNA repair gene mutations result from the accumulation of DNA damage?The oxidatively induced DNA lesions 8,5'-cyclo-2'-deoxyadenosine and 8-hydroxy-2'-deoxyadenosine are strongly resistant to acid-induced hydrolysis of the glycosidic bondNew applications of the Comet assay: Comet-FISH and transcription-coupled DNA repair.The bright and the dark sides of DNA repair in stem cells.Genetic instability in human embryonic stem cells: prospects and caveats.Transcription-coupled repair: an update.Mechanistic insights into transcription coupled DNA repair.Transcription-induced CAG repeat contraction in human cells is mediated in part by transcription-coupled nucleotide excision repair.Emerging links between premature ageing and defective DNA repair.Attenuated nucleotide excision repair leads to mutagenesis in cancer cells.Deficient DNA damage response and cell cycle checkpoints lead to accumulation of point mutations in human embryonic stem cells.MutationalPatterns: comprehensive genome-wide analysis of mutational processes.Genomic Instability and Cancer.
P2860
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P2860
description
2006 nî lūn-bûn
@nan
2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Transcription domain-associated repair in human cells.
@ast
Transcription domain-associated repair in human cells.
@en
type
label
Transcription domain-associated repair in human cells.
@ast
Transcription domain-associated repair in human cells.
@en
prefLabel
Transcription domain-associated repair in human cells.
@ast
Transcription domain-associated repair in human cells.
@en
P2860
P356
P1476
Transcription domain-associated repair in human cells.
@en
P2093
Philip C Hanawalt
Thierry P Nouspikel
P2860
P304
P356
10.1128/MCB.01263-06
P407
P577
2006-10-02T00:00:00Z