Terminally differentiated muscle cells are defective in base excision DNA repair and hypersensitive to oxygen injury.
about
Defective DNA ligation during short-patch single-strand break repair in ataxia oculomotor apraxia 1The Response to Oxidative DNA Damage in Neurons: Mechanisms and DiseaseBalancing self-renewal against genome preservation in stem cells: How do they manage to have the cake and eat it too?Base excision repairp53 coordinates base excision repair to prevent genomic instabilityAge- and tissue-specific changes in mitochondrial and nuclear DNA base excision repair activity in mice: Susceptibility of skeletal muscles to oxidative injury.Cyclin-C-dependent cell-cycle entry is required for activation of non-homologous end joining DNA repair in postmitotic neuronsTelomeres shorten in response to oxidative stress in mouse skeletal muscle fibersRedox or death: checking on fetal myogenesis.DNA repair deficiency in neurodegeneration.DNA polymerase beta-catalyzed-PCNA independent long patch base excision repair synthesis: a mechanism for repair of oxidatively damaged DNA ends in post-mitotic brain.DNA repair kinetics in SCID mice Sertoli cells and DNA-PKcs-deficient mouse embryonic fibroblasts.Deficiency in repair of the mitochondrial genome sensitizes proliferating myoblasts to oxidative damageCreating a monomeric endonuclease TALE-I-SceI with high specificity and low genotoxicity in human cells.Irreparable complex DNA double-strand breaks induce chromosome breakage in organotypic three-dimensional human lung epithelial cell cultureHuman monocytes are severely impaired in base and DNA double-strand break repair that renders them vulnerable to oxidative stressSlow mitochondrial repair of 5'-AMP renders mtDNA susceptible to damage in APTX deficient cells.The nucleotide sequence, DNA damage location, and protein stoichiometry influence the base excision repair outcome at CAG/CTG repeatsQuiescent keratocytes fail to repair MMC induced DNA damage leading to the long-term inhibition of myofibroblast differentiation and wound healingActivities of DNA base excision repair enzymes in liver and brain correlate with body mass, but not lifespan.Alcohol-induced one-carbon metabolism impairment promotes dysfunction of DNA base excision repair in adult brain.Base excision repair, aging and health span.XRCC1 protects against the lethality of induced oxidative DNA damage in nondividing neural cellsBroad DNA repair responses in neural injury are associated with activation of the IL-6 pathway in cholesterol-fed rabbits.miR-638 suppresses DNA damage repair by targeting SMC1A expression in terminally differentiated cellsCheckpoint kinase phosphorylation in response to endogenous oxidative DNA damage in repair-deficient stationary-phase Saccharomyces cerevisiaeDNA damage-activated ABL-MyoD signaling contributes to DNA repair in skeletal myoblasts.Myostatin induces DNA damage in skeletal muscle of streptozotocin-induced type 1 diabetic mice.The bright and the dark sides of DNA repair in stem cells.The role of DNA base excision repair in brain homeostasis and disease.The impact of base excision DNA repair in age-related neurodegenerative diseasesHIRA deficiency in muscle fibers causes hypertrophy and susceptibility to oxidative stress.mESC-based in vitro differentiation models to study vascular response and functionality following genotoxic insults.Risky repair: DNA-protein crosslinks formed by mitochondrial base excision DNA repair enzymes acting on free radical lesions.Modulation of DNA base excision repair during neuronal differentiation.Coordination of cell cycle, DNA repair and muscle gene expression in myoblasts exposed to genotoxic stress.DNA Damage Response and DNA Repair in Skeletal Myocytes From a Mouse Model of Spinal Muscular Atrophy.Temporal activation of XRCC1-mediated DNA repair is essential for muscle differentiation.c-Fos-dependent miR-22 targets MDC1 and regulates DNA repair in terminally differentiated cells.DNA damage response by single-strand breaks in terminally differentiated muscle cells and the control of muscle integrity
P2860
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P2860
Terminally differentiated muscle cells are defective in base excision DNA repair and hypersensitive to oxygen injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Terminally differentiated musc ...... persensitive to oxygen injury.
@ast
Terminally differentiated musc ...... persensitive to oxygen injury.
@en
type
label
Terminally differentiated musc ...... persensitive to oxygen injury.
@ast
Terminally differentiated musc ...... persensitive to oxygen injury.
@en
prefLabel
Terminally differentiated musc ...... persensitive to oxygen injury.
@ast
Terminally differentiated musc ...... persensitive to oxygen injury.
@en
P2093
P2860
P50
P356
P1476
Terminally differentiated musc ...... ypersensitive to oxygen injury
@en
P2093
Bruce Demple
Eugenia Dogliotti
Laura Narciso
Mathilde Frechet
Paolo Degan
Pingfang Liu
P2860
P304
17010-17015
P356
10.1073/PNAS.0701743104
P407
P577
2007-10-16T00:00:00Z