Rb and N-ras function together to control differentiation in the mouse. - Wikidata - wikimedia - TriplyDB

Rb and N-ras function together to control differentiation in the mouse.

Rb and N-ras function together to control differentiation in the mouse.

http://www.wikidata.org/entity/Q35161806

about

Inhibition of oncogenic transformation by mammalian Lin-9, a pRB-associated protein Immortalization of mouse myogenic cells can occur without loss of p16INK4a, p19ARF, or p53 and is accelerated by inactivation of Bax Retinoblastoma tumor suppressor functions shared by stem cell and cancer cell strategies Twists in views on RB functions in cellular signaling, metabolism and stem cells Critical role of the Rb family in myoblast survival and fusion Genetic interaction between Rb and K-ras in the control of differentiation and tumor suppression The retinoblastoma gene pathway regulates the postmitotic state of hair cells of the mouse inner ear.Perturbation biology: inferring signaling networks in cellular systems The retinoblastoma tumor-suppressor gene, the exception that proves the rule Transient inactivation of Rb and ARF yields regenerative cells from postmitotic mammalian muscle Retinoblastoma protein and MyoD function together to effect the repression of Fra-1 and in turn cyclin D1 during terminal cell cycle arrest associated with myogenesis.An E2F binding-deficient Rb1 protein partially rescues developmental defects associated with Rb1 nullizygosity.Wild-type NRas and KRas perform distinct functions during transformation.Let-7a regulates mammosphere formation capacity through Ras/NF-κB and Ras/MAPK/ERK pathway in breast cancer stem cells Rb is required for progression through myogenic differentiation but not maintenance of terminal differentiation.ATM mediates pRB function to control DNMT1 protein stability and DNA methylation.Posttranslational modifications of the retinoblastoma tumor suppressor protein as determinants of function.A gradient of epidermal growth factor receptor signaling determines the sensitivity of rbf1 mutant cells to E2F-dependent apoptosis.Rb/E2F1 regulates the innate immune receptor Toll-like receptor 3 in epithelial cells.The retinoblastoma gene Rb and its family member p130 suppress lung adenocarcinoma induced by oncogenic K-Ras.Fascin Is Critical for the Maintenance of Breast Cancer Stem Cell Pool Predominantly via the Activation of the Notch Self-Renewal Pathway.Differential effects of c-Ras upon transformation, adipocytic differentiation, and apoptosis mediated by the simian virus 40 large tumor antigen.pRb-dependent cyclin D3 protein stabilization is required for myogenic differentiation.

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P2860

Rb and N-ras function together to control differentiation in the mouse.

http://www.wikidata.org/entity/Q35161806

description

2003 nî lūn-bûn

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2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2003年の論文

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2003年論文

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2003年論文

@zh-hant

2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

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name

Rb and N-ras function together to control differentiation in the mouse.

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Rb and N-ras function together to control differentiation in the mouse.

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type

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Rb and N-ras function together to control differentiation in the mouse.

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Rb and N-ras function together to control differentiation in the mouse.

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Rb and N-ras function together to control differentiation in the mouse.

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Rb and N-ras function together to control differentiation in the mouse.

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MCB.23.15.5256-5268.2003

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Molecular and Cellular Biology

P1476

Rb and N-ras function together to control differentiation in the mouse.

@en

P2093

Bernardo Contreras

http://www.w3.org/2001/XMLSchema#string

Chiaki Takahashi

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Kwang Youl Lee

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Makoto Noda

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Mark E Ewen

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Merav Socolovsky

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Raju Kucherlapati

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Roderick T Bronson

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Tyler Jacks

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P2860

The retinoblastoma protein and cell cycle control

E2F4 and E2F5 play an essential role in pocket protein-mediated G1 control

Ras signalling is required for inactivation of the tumour suppressor pRb cell-cycle control protein

Characterization of a human MSX-2 cDNA and its fragment isolated as a transformation suppressor gene against v-Ki-ras oncogene

Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein

Targeted disruption of the glucocorticoid receptor gene blocks adrenergic chromaffin cell development and severely retards lung maturation

Targeted deletion of the H-ras gene decreases tumor formation in mouse skin carcinogenesis

Dual roles of the retinoblastoma protein in cell cycle regulation and neuron differentiation

Phosphorylation at threonine-235 by a ras-dependent mitogen-activated protein kinase cascade is essential for transcription factor NF-IL6

ras oncogenes in human cancer: a review

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5256-5268

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10.1128/MCB.23.15.5256-5268.2003

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15

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23

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