Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease - Wikidata - wikimedia - TriplyDB

Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease

Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease

http://www.wikidata.org/entity/Q35167904

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Tau Oligomers: The Toxic Player at Synapses in Alzheimer's Disease Kynurenine 3-Monooxygenase: An Influential Mediator of Neuropathology Does neuroinflammation turn on the flame in Alzheimer's disease? Focus on astrocytes Tau-tubulin kinase Neuropathological alterations in Alzheimer disease Neuroinflammation in Alzheimer's disease Depletion of microglia and inhibition of exosome synthesis halt tau propagation.Neuropsychiatric Disturbances in Alzheimer's Disease: What Have We Learned from Neuropathological Studies?Friend or foe: the dichotomous impact of T cells on neuro-de/re-generation during aging Systems analysis of human brain gene expression: mechanisms for HIV-associated neurocognitive impairment and common pathways with Alzheimer's disease ³H-deprenyl and ³H-PIB autoradiography show different laminar distributions of astroglia and fibrillar β-amyloid in Alzheimer brain Early and progressive deficit of neuronal activity patterns in a model of local amyloid pathology in mouse prefrontal cortex Alzheimer's Disease Cerebrospinal Fluid and Neuroimaging Biomarkers: Diagnostic Accuracy and Relationship to Drug Efficacy.Noninvasive and targeted delivery of therapeutics to the brain using focused ultrasound.Future Directions in Imaging Neurodegeneration.The intersection of amyloid beta and tau at synapses in Alzheimer's disease Neuroinflammation and neuronal loss precede Aβ plaque deposition in the hAPP-J20 mouse model of Alzheimer's disease.A reduced astrocyte response to β-amyloid plaques in the ageing brain associates with cognitive impairment.Apolipoprotein E4 effects in Alzheimer's disease are mediated by synaptotoxic oligomeric amyloid-β Stable size distribution of amyloid plaques over the course of Alzheimer disease.Evidence for miR-181 involvement in neuroinflammatory responses of astrocytes.Statins induce insulin-degrading enzyme secretion from astrocytes via an autophagy-based unconventional secretory pathway.Plaque-Associated Local Toxicity Increases over the Clinical Course of Alzheimer Disease.Presenilin-1 adopts pathogenic conformation in normal aging and in sporadic Alzheimer's disease.Abnormal tau induces cognitive impairment through two different mechanisms: synaptic dysfunction and neuronal loss LPS-induced Murine Neuroinflammation Model: Main Features and Suitability for Pre-clinical Assessment of Nutraceuticals Differential relationships of reactive astrocytes and microglia to fibrillar amyloid deposits in Alzheimer disease.A phenotypic change but not proliferation underlies glial responses in Alzheimer disease Alzheimer's disease risk gene CD33 inhibits microglial uptake of amyloid beta.Dissecting phenotypic traits linked to human resilience to Alzheimer's pathology.Regional and Gender Study of Neuronal Density in Brain during Aging and in Alzheimer's Disease.Amyloid-β 1-24 C-terminal truncated fragment promotes amyloid-β 1-42 aggregate formation in the healthy brain.Amyloid β peptide-mediated neurotoxicity is attenuated by the proliferating microglia more potently than by the quiescent phenotype.Soluble phospho-tau from Alzheimer's disease hippocampus drives microglial degeneration.CB₂ receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease.Microglia convert aggregated amyloid-β into neurotoxic forms through the shedding of microvesicles.CB2 receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease.Examination of the clinicopathologic continuum of Alzheimer disease in the autopsy cohort of the National Alzheimer Coordinating Center.Early changes in CSF sTREM2 in dominantly inherited Alzheimer's disease occur after amyloid deposition and neuronal injury.Oxidative stress in Alzheimer's disease: Primary villain or physiological by-product?

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Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease

http://www.wikidata.org/entity/Q35167904

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2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Reactive glia not only associa ...... tangles in Alzheimer's disease

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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J.AJPATH.2011.05.047

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The American Journal of Pathology

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Reactive glia not only associa ...... tangles in Alzheimer's disease

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P2093

Alberto Serrano-Pozo

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John H Growdon

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Matthew L Mielke

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Matthew P Frosch

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Rebecca A Betensky

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Teresa Gómez-Isla

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P2860

Selective induction of astrocytic gliosis generates deficits in neuronal inhibition

Effect of non-steroidal anti-inflammatory drugs on risk of Alzheimer's disease: systematic review and meta-analysis of observational studies

Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

Neuropathological stageing of Alzheimer-related changes

Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model

Nonsteroidal anti-inflammatory drugs are associated with increased neuritic plaques

Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment

Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade

Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease

Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis

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51510061

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Alzheimer's disease

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