Pan-PPAR modulation effectively protects APP/PS1 mice from amyloid deposition and cognitive deficits - Wikidata - wikimedia - TriplyDB

Pan-PPAR modulation effectively protects APP/PS1 mice from amyloid deposition and cognitive deficits

Pan-PPAR modulation effectively protects APP/PS1 mice from amyloid deposition and cognitive deficits

http://www.wikidata.org/entity/Q35176258

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Insulin resistance in Alzheimer's disease.New Insights into Epigenetic and Pharmacological Regulation of Amyloid-Degrading Enzymes.Therapeutic potential of nuclear receptor agonists in Alzheimer's disease.Personalized genetics of the cholinergic blockade of neuroinflammation.Targeting neuroinflammation in Alzheimer's disease: evidence for NSAIDs and novel therapeutics.Calycosin improves cognitive function in a transgenic mouse model of Alzheimer's disease by activating the protein kinase C pathway.Microglia and astrocytes in Alzheimer's disease: implications for therapy.Comparative transcriptomics of choroid plexus in Alzheimer's disease, frontotemporal dementia and Huntington's disease: implications for CSF homeostasis.

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P2860

Pan-PPAR modulation effectively protects APP/PS1 mice from amyloid deposition and cognitive deficits

http://www.wikidata.org/entity/Q35176258

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Molecular Neurobiology

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Pan-PPAR modulation effectivel ...... osition and cognitive deficits

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Daisy Axt

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Dean W Hum

http://www.w3.org/2001/XMLSchema#string

Markus P Kummer

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Mathieu Dubernet

http://www.w3.org/2001/XMLSchema#string

Michael T Heneka

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Rafael Schwarzenberger

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Robert Walczak

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Sakina Sayah-Jeanne

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Stephanie Schwartz

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P2860

Alzheimer's Disease

Amyloid-beta-induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks

ApoE-directed therapeutics rapidly clear β-amyloid and reverse deficits in AD mouse models

Focal glial activation coincides with increased BACE1 activation and precedes amyloid plaque deposition in APP[V717I] transgenic mice.

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Peroxisome proliferator-activated receptor delta (PPARdelta) activation protects H9c2 cardiomyoblasts from oxidative stress-induced apoptosis

Decreased clearance of CNS beta-amyloid in Alzheimer's disease

PPAR-gamma agonists inhibit production of monocyte inflammatory cytokines

The peroxisome proliferator-activated receptor-gamma is a negative regulator of macrophage activation

Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers

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s12035-014-8743-4

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661-671

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scholarly article

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10.1007/S12035-014-8743-4

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2

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51

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24838579

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