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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

Cellular FLICE-inhibitory protein: an attractive therapeutic target?

http://www.wikidata.org/entity/Q35186676

about

Elevated NF-kappaB responses and FLIP levels in leukemic but not normal lymphocytes: reduction by salicylate allows TNF-induced apoptosis Lesser-Known Molecules in Ovarian Carcinogenesis c-FLIP, a master anti-apoptotic regulator The heme oxygenase-1 and c-FLIP in acute myeloid leukemias: two non-redundant but mutually exclusive cellular safeguards protecting cells against TNF-induced cell death?Fas-associated protein with death domain (FADD)-independent recruitment of c-FLIPL to death receptor 5.Endogenous Bak inhibitors Mcl-1 and Bcl-xL: differential impact on TRAIL resistance in Bax-deficient carcinoma.Cellular stress responses: cell survival and cell death FLIP protects against hypoxia/reoxygenation-induced endothelial cell apoptosis by inhibiting Bax activation Differential inhibition of TRAIL-mediated DR5-DISC formation by decoy receptors 1 and 2.Expression of cellular FLICE inhibitory proteins (cFLIP) in normal and traumatic murine and human cerebral cortex TRAIL in cancer therapy: present and future challenges.Roles of c-FLIP in Apoptosis, Necroptosis, and Autophagy.Fas determines differential fates of resident and recruited macrophages during resolution of acute lung injury.Regulating TRAIL receptor-induced cell death at the membrane : a deadly discussion Targeting the Anti-Apoptotic Protein c-FLIP for Cancer Therapy.cFLIP is critical for oligodendrocyte protection from inflammation Modulation of apoptosis as a target for liver disease.Hyperthermia restores apoptosis induced by death receptors through aggregation-induced c-FLIP cytosolic depletion.Computational systems biology in cancer: modeling methods and applications.Valproate activates the Notch3/c-FLIP signaling cascade: a strategy to attenuate white matter hyperintensities in bipolar disorder in late life?Hyperoxia sensing: from molecular mechanisms to significance in disease.Cellular FLICE-Inhibitory Protein Regulates Tissue Homeostasis.Type I interferons induce apoptosis by balancing cFLIP and caspase-8 independent of death ligands.Inhibition of latent membrane protein 1 impairs the growth and tumorigenesis of latency II Epstein-Barr virus-transformed T cells.Regulation of cell death in cancer-possible implications for immunotherapy.CD8+ T cells use TRAIL to restrict West Nile virus pathogenesis by controlling infection in neurons.Mathematical modeling reveals threshold mechanism in CD95-induced apoptosis.Simultaneous induction of apoptosis and necroptosis by Tanshinone IIA in human hepatocellular carcinoma HepG2 cells.Tumor necrosis factor (TNF) receptor-associated factor 7 is required for TNFα-induced Jun NH2-terminal kinase activation and promotes cell death by regulating polyubiquitination and lysosomal degradation of c-FLIP protein.Evasion of apoptosis as a cellular stress response in cancer.Betulinic Acid for cancer treatment and prevention.Release of c-FLIP brake selectively sensitizes human cancer cells to TLR3-mediated apoptosis

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P2860

Cellular FLICE-inhibitory protein: an attractive therapeutic target?

http://www.wikidata.org/entity/Q35186676

description

2003 nî lūn-bûn

@nan

2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի օգոստոսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Expert Opinion on Therapeutic Targets

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Cellular FLICE-inhibitory protein: an attractive therapeutic target?

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Olivier Micheau

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P2860

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

Mutations in the human homologue of mouse dl cause autosomal recessive and dominant hypohidrotic ectodermal dysplasia

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

Amyloid beta binds trimers as well as monomers of the 75-kDa neurotrophin receptor and activates receptor signaling

Expression and structure of the human NGF receptor

Soluble forms of tumor necrosis factor receptors (TNF-Rs). The cDNA for the type I TNF-R, cloned using amino acid sequence data of its soluble form, encodes both the cell surface and a soluble form of the receptor

Apo-3, a new member of the tumor necrosis factor receptor family, contains a death domain and activates apoptosis and NF-kappa B

Signal transduction by DR3, a death domain-containing receptor related to TNFR-1 and CD95

LARD: a new lymphoid-specific death domain containing receptor regulated by alternative pre-mRNA splicing

Casper is a FADD- and caspase-related inducer of apoptosis

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559-573

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scholarly article

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10.1517/14728222.7.4.559

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