Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration.
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Immature B Cell Egress from Bone Marrow Is SOCS3 IndependentThe transcription factor EPAS1 links DOCK8 deficiency to atopic skin inflammation via IL-31 inductionDOCK8 regulates signal transduction events to control immunityEditing the Mouse Genome Using the CRISPR-Cas9 SystemRecent Advances in DOCK8 Immunodeficiency Syndrome.Skin Immunity to Candida albicans.The pathogenic role of the inflammasome in neurodegenerative diseases.Dendritic cell migration in health and disease.Heme drives hemolysis-induced susceptibility to infection via disruption of phagocyte functions.Frontline Science: Coincidental null mutation of Csf2rα in a colony of PI3Kγ-/- mice causes alveolar macrophage deficiency and fatal respiratory viral infection.Epidermal NLRP10 contributes to contact hypersensitivity responses in mice.CCDC88B is required for pathogenesis of inflammatory bowel diseaseNLRP10 Enhances CD4+ T-Cell-Mediated IFNγ Response via Regulation of Dendritic Cell-Derived IL-12 Release.Epidermal cornification is preceded by the expression of a keratinocyte-specific set of pyroptosis-related genes.Danger of frustrated sensors: Role of Toll-like receptors and NOD-like receptors in aseptic and septic inflammations around total hip replacements.Migratory CD11b+ conventional dendritic cells induce T follicular helper cell-dependent antibody responses.An Anti-Inflammatory Role for NLRP10 in Murine Cutaneous Leishmaniasis.Pattern of cardiotoxin-induced muscle remodeling in distinct TLR-4 deficient mouse strains.Mast cell heterogeneity underlies different manifestations of food allergy in mice.DOCK8 Protein Regulates Macrophage Migration through Cdc42 Protein Activation and LRAP35a Protein Interaction.MyD88-dependent dendritic and epithelial cell crosstalk orchestrates immune responses to allergens.PYNOD reduces microglial inflammation and consequent neurotoxicity upon lipopolysaccharides stimulation.CRISPR therapeutic tools for complex genetic disorders and cancer (Review).Determination of T Follicular Helper Cell Fate by Dendritic Cells
P2860
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P2860
Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration.
description
2015 nî lūn-bûn
@nan
2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@ast
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@en
type
label
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@ast
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@en
prefLabel
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@ast
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@en
P2093
P2860
P50
P356
P1476
Coincidental loss of DOCK8 fun ...... tive dendritic cell migration.
@en
P2093
Adam Williams
André C Müller
Anne Marie Rhebergen
Antonia Gallman
Anuj Srivastava
Arpita Singh
Fayyaz S Sutterwala
James D Gorham
Jorge Henao-Mejia
P2860
P304
P356
10.1073/PNAS.1501554112
P407
P50
P577
2015-02-23T00:00:00Z