PYNOD reduces microglial inflammation and consequent neurotoxicity upon lipopolysaccharides stimulation.

about

PYNOD reduces microglial inflammation and consequent neurotoxicity upon lipopolysaccharides stimulation.

Item

http://www.wikidata.org/entity/Q55347487

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@en

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@nl

type

Item

label

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@en

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@nl

prefLabel

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@en

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@nl

P1476

PYNOD reduces microglial infla ...... popolysaccharides stimulation.

@en

P2093

Chaofeng Hu

http://www.w3.org/2001/XMLSchema#string

Daxiang Lu

http://www.w3.org/2001/XMLSchema#string

Qi Zeng

http://www.w3.org/2001/XMLSchema#string

Renbin Qi

http://www.w3.org/2001/XMLSchema#string

P2860

NF-kappaB Signaling Pathways in Neurological Inflammation: A Mini Review

Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease

Immediate early response of the circadian polyA ribonuclease nocturnin to two extracellular stimuli.

NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells

Microglia induce motor neuron death via the classical NF-κB pathway in amyotrophic lateral sclerosis.

Anti-inflammatory activity of PYNOD and its mechanism in humans and mice.

PYNOD, a novel Apaf-1/CED4-like protein is an inhibitor of ASC and caspase-1.

PYPAF3, a PYRIN-containing APAF-1-like protein, is a feedback regulator of caspase-1-dependent interleukin-1beta secretion.

NOD1 and NOD2: signaling, host defense, and inflammatory disease.

Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration.

P304

5337-5343

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3892/ETM.2018.6108

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-04-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29904414

http://www.w3.org/2001/XMLSchema#string

P921

microglia

neurotoxicity

lipopolysaccharide

P932

5996706

http://www.w3.org/2001/XMLSchema#string