Loss of SOD3 (EcSOD) Expression Promotes an Aggressive Phenotype in Human Pancreatic Ductal Adenocarcinoma.
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Extracellular Superoxide Dismutase: Growth Promoter or Tumor Suppressor?Disulfiram anti-cancer efficacy without copper overload is enhanced by extracellular H2O2 generation: antagonism by tetrathiomolybdate.Histone deacetylation contributes to low extracellular superoxide dismutase expression in human idiopathic pulmonary arterial hypertension.Expanding roles of superoxide dismutases in cell regulation and cancerThe role of hypoxia in pancreatic cancer: a potential therapeutic target?NO• and Pancreatic Cancer: A Complex Interaction with Therapeutic Potential.Unraveling new functions of superoxide dismutase using yeast model system: Beyond its conventional role in superoxide radical scavenging.Targeting reactive oxygen species in development and progression of pancreatic cancer.Paradoxical roles of dual oxidases in cancer biology.The Strong Cell-based Hydrogen Peroxide Generation Triggered by Cold Atmospheric PlasmaSuperoxide Dismutases in Pancreatic Cancer.On the Origin of Superoxide Dismutase: An Evolutionary Perspective of Superoxide-Mediated Redox Signaling.Redox Regulation of the Superoxide Dismutases SOD3 and SOD2 in the Pulmonary Circulation.Extracellular superoxide dismutase and its role in cancer.Extracellular superoxide dismutase inhibits hepatocyte growth factor-mediated breast cancer-fibroblast interactions.Copper chaperone antioxidant-1, Atox-1, is involved in the induction of SOD3 in THP-1 cells.Extracellular redox state shift: A novel approach to target prostate cancer invasion.SOD3 improves the tumor response to chemotherapy by stabilizing endothelial HIF-2α.Ten-eleven translocation 1 functions as a mediator of SOD3 expression in human lung cancer A549 cells.Identification of differentially expressed proteins in the gastric mucosal atypical hyperplasia tissue microenvironment.The manganese(III) porphyrin MnTnHex-2-PyP5+ modulates intracellular ROS and breast cancer cell migration: impact in doxorubicin-treated cellsFenton reactions drive nucleotide and ATP syntheses in cancer
P2860
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P2860
Loss of SOD3 (EcSOD) Expression Promotes an Aggressive Phenotype in Human Pancreatic Ductal Adenocarcinoma.
description
2015 nî lūn-bûn
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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2015 թվականի հունվարին հրատարակված գիտական հոդված
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@ast
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@en
type
label
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@ast
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@en
prefLabel
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@ast
Loss of SOD3 (EcSOD) Expressio ...... creatic Ductal Adenocarcinoma.
@en
P2093
P2860
P50
P1476
Loss of SOD3 (EcSOD) Expressio ...... ncreatic Ductal Adenocarcinoma
@en
P2093
Anna M Button
Brenda Y Hernandez
Brett A Wagner
Brianne R O'Leary
Charles F Lynch
James J Mezhir
Jeffery Keene
Robert A Beardsley
Sean Altekruse
Wendy Cozen
P2860
P304
P356
10.1158/1078-0432.CCR-14-1959
P407
P50
P577
2015-01-29T00:00:00Z