Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle. - Wikidata - wikimedia - TriplyDB

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

http://www.wikidata.org/entity/Q35286959

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Oxidative Stress-Mediated Skeletal Muscle Degeneration: Molecules, Mechanisms, and Therapies Constitutive expression of Yes-associated protein (Yap) in adult skeletal muscle fibres induces muscle atrophy and myopathy Disrupted autophagy undermines skeletal muscle adaptation and integrity From Nutrient to MicroRNA: a Novel Insight into Cell Signaling Involved in Skeletal Muscle Development and Disease Reduced IGF signaling prevents muscle cell death in a Caenorhabditis elegans model of muscular dystrophy Rapamycin nanoparticles target defective autophagy in muscular dystrophy to enhance both strength and cardiac function Src-dependent impairment of autophagy by oxidative stress in a mouse model of Duchenne muscular dystrophy Skeletal muscle homeostasis in duchenne muscular dystrophy: modulating autophagy as a promising therapeutic strategy.The intriguing regulators of muscle mass in sarcopenia and muscular dystrophy.PKC theta ablation improves healing in a mouse model of muscular dystrophy.Autophagy is Impaired in the Tibialis Anterior of Dystrophin Null Mice A novel mechanism of autophagic cell death in dystrophic muscle regulated by P2RX7 receptor large-pore formation and HSP90.Effect of rapamycin on immunity induced by vector-mediated dystrophin expression in mdx skeletal muscle Of mice and men: the benefits of caloric restriction, exercise, and mimetics.Adeno-associated virus serotype 8 (AAV8) delivery of recombinant A20 to skeletal muscle reduces pathological activation of nuclear factor (NF)-κB in muscle of mdx mice.Rapamycin reverses elevated mTORC1 signaling in lamin A/C-deficient mice, rescues cardiac and skeletal muscle function, and extends survival.Regulatory T cells suppress muscle inflammation and injury in muscular dystrophy.Four-week rapamycin treatment improves muscular dystrophy in a fukutin-deficient mouse model of dystroglycanopathy Controlled release formulations of IL-2, TGF-β1 and rapamycin for the induction of regulatory T cells.Tissue Tregs.Adipose-derived stem cells enhance myogenic differentiation in the mdx mouse model of muscular dystrophy via paracrine signaling."Get the Balance Right": Pathological Significance of Autophagy Perturbation in Neuromuscular Disorders S1P lyase in skeletal muscle regeneration and satellite cell activation: exposing the hidden lyase The Skeletal Muscle Environment and Its Role in Immunity and Tolerance to AAV Vector-Mediated Gene Transfer.Immune-mediated pathology in Duchenne muscular dystrophy.Myofibrillar Myopathies: New Perspectives from Animal Models to Potential Therapeutic Approaches.Evolving paradigms in clinical pharmacology and therapeutics for the treatment of Duchenne muscular dystrophy.Autophagy as a new therapeutic target in Duchenne muscular dystrophy From innate to adaptive immune response in muscular dystrophies and skeletal muscle regeneration: the role of lymphocytes RhoA/ROCK inhibition improves the beneficial effects of glucocorticoid treatment in dystrophic muscle: implications for stem cell depletion.

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P2860

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

http://www.wikidata.org/entity/Q35286959

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@ast

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@en

type

label

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@ast

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@en

prefLabel

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@ast

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@en

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MOLMED.2010.00256

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Molecular Medicine

P1476

Rapamycin ameliorates dystrophic phenotype in mdx mouse skeletal muscle.

@en

P2093

Paula R Clemens

http://www.w3.org/2001/XMLSchema#string

Saman Eghtesad

http://www.w3.org/2001/XMLSchema#string

Siddharth Jhunjhunwala

http://www.w3.org/2001/XMLSchema#string

Steven R Little

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P2860

Rapamycin fed late in life extends lifespan in genetically heterogeneous mice

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

Population frequencies of inherited neuromuscular diseases--a world survey

X chromosome-linked muscular dystrophy (mdx) in the mouse

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy.

Immunoregulatory functions of mTOR inhibition.

The complexes of mammalian target of rapamycin.

Do immune cells promote the pathology of dystrophin-deficient myopathies?

Protein turnover in cardiac cell growth and survival.

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molmed.2010.00256

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917-924

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