Junin virus infects mouse cells and induces innate immune responses. - Wikidata - wikimedia - TriplyDB

Junin virus infects mouse cells and induces innate immune responses.

Junin virus infects mouse cells and induces innate immune responses.

http://www.wikidata.org/entity/Q35363337

about

Junín virus pathogenesis and virus replication Calcium Regulation of Hemorrhagic Fever Virus Budding: Mechanistic Implications for Host-Oriented Therapeutic Intervention Potent inhibition of Junín virus infection by interferon in murine cells.Utilization of human DC-SIGN and L-SIGN for entry and infection of host cells by the New World arenavirus, Junín virus.Toll-like receptor 2-mediated innate immune responses against Junín virus in mice lead to antiviral adaptive immune responses during systemic infection and do not affect viral replication in the brain Junín virus infection activates the type I interferon pathway in a RIG-I-dependent manner A systems biology starter kit for arenaviruses.Sorting of small infectious virus particles by flow virometry reveals distinct infectivity profiles.Contemplating the Importance of Toll-like Receptors I and II Regarding Human Viral Pathogenesis.Highly Pathogenic New World and Old World Human Arenaviruses Induce Distinct Interferon Responses in Human Cells.Pathogenic Old World arenaviruses inhibit TLR2/Mal-dependent proinflammatory cytokines in vitro Computational and Functional Analysis of the Virus-Receptor Interface Reveals Host Range Trade-Offs in New World Arenaviruses.Evaluation of Lassa virus vaccine immunogenicity in a CBA/J-ML29 mouse model Nonhuman transferrin receptor 1 is an efficient cell entry receptor for Ocozocoautla de Espinosa virus.Innate immune response to arenaviral infection: a focus on the highly pathogenic New World hemorrhagic arenaviruses.MyD88-dependent immunity to a natural model of vaccinia virus infection does not involve Toll-like receptor 2 A host-oriented inhibitor of Junin Argentine hemorrhagic fever virus egress.Host cell factors as antiviral targets in arenavirus infection.Arenavirus evasion of host anti-viral responses.Highly Pathogenic New World Arenavirus Infection Activates the Pattern Recognition Receptor Protein Kinase R without Attenuating Virus Replication in Human Cells.AIM2-Like Receptors Positively and Negatively Regulate the Interferon Response Induced by Cytosolic DNA siRNA screen for genes that affect Junín virus entry uncovers voltage-gated calcium channels as a therapeutic target.Involvement of Pro-Inflammatory Macrophages in Liver Pathology of Pirital Virus-Infected Syrian Hamsters.Review of Biology and Replication

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P2860

Junin virus infects mouse cells and induces innate immune responses.

http://www.wikidata.org/entity/Q35363337

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Junin virus infects mouse cells and induces innate immune responses.

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Junin virus infects mouse cells and induces innate immune responses.

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type

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Junin virus infects mouse cells and induces innate immune responses.

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Junin virus infects mouse cells and induces innate immune responses.

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prefLabel

Junin virus infects mouse cells and induces innate immune responses.

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Junin virus infects mouse cells and induces innate immune responses.

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P1433

Journal of Virology

P1476

Junin virus infects mouse cells and induces innate immune responses

@en

P2093

Christian D Cuevas

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Enxiu Wang

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Madakasira Lavanya

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P2860

Z proteins of New World arenaviruses bind RIG-I and interfere with type I interferon induction

Tracking Virus-Specific CD4+ T Cells during and after Acute Hepatitis C Virus Infection

Host-Species Transferrin Receptor 1 Orthologs Are Cellular Receptors for Nonpathogenic New World Clade B Arenaviruses

Differences in tropism and pH dependence for glycoproteins from the Clade B1 arenaviruses: Implications for receptor usage and pathogenicity

Structural basis for receptor recognition by New World hemorrhagic fever arenaviruses

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

The interferon regulatory factor, IRF5, is a central mediator of toll-like receptor 7 signaling

Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

Innate antiviral responses by means of TLR7-mediated recognition of single-stranded RNA

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11058-11068

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scholarly article

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10.1128/JVI.05304-11

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21

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85

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2011-08-31T00:00:00Z

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21880772

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3194972

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