Helicobacter pylori requires an acidic environment to survive in the presence of urea.
about
Effect of Helicobacter pylori eradication on the natural history of duodenal ulcer diseaseAspirin inhibits the growth of Helicobacter pylori and enhances its susceptibility to antimicrobial agentsIdentification of virulence genes of Helicobacter pylori by random insertion mutagenesisDetermination of the infectious dose of Helicobacter pylori during primary and secondary infection in rhesus monkeys (Macaca mulatta).Cytoplasmic histidine kinase (HP0244)-regulated assembly of urease with UreI, a channel for urea and its metabolites, CO2, NH3, and NH4(+), is necessary for acid survival of Helicobacter pylori.Synthesis and activity of Helicobacter pylori urease and catalase at low pHIn vitro evaluation of the role of antibodies against Helicobacter pylori in inhibiting adherence of the organism to gastric cells.Factors that mediate colonization of the human stomach by Helicobacter pylori.Helicobacter pylori rocF is required for arginase activity and acid protection in vitro but is not essential for colonization of mice or for urease activity.Metabolism and genetics of Helicobacter pylori: the genome era.Antagonistic activity against Helicobacter infection in vitro and in vivo by the human Lactobacillus acidophilus strain LBTranscriptional regulation of the Streptococcus salivarius 57.I urease operon.Pretreatment with urea-hydrochloric acid enhances the isolation of Helicobacter pylori from contaminated specimensEmergence of diverse Helicobacter species in the pathogenesis of gastric and enterohepatic diseases.Development of a selective medium for isolation of Helicobacter pylori from cattle and beef samples.Selection for urease activity during Helicobacter pylori infection of rhesus macaques (Macaca mulatta).Protein glycosylation in Helicobacter pylori: beyond the flagellins?Ammonium metabolism enzymes aid Helicobacter pylori acid resistance.In vivo behavior of a Helicobacter pylori SS1 nixA mutant with reduced urease activityThe Helicobacter pylori homologue of the ferric uptake regulator is involved in acid resistance.NikR mediates nickel-responsive transcriptional induction of urease expression in Helicobacter pylori.A cis-encoded antisense small RNA regulated by the HP0165-HP0166 two-component system controls expression of ureB in Helicobacter pylori.Helicobacter pylori 5'ureB-sRNA, a cis-encoded antisense small RNA, negatively regulates ureAB expression by transcription terminationConstruction and characterization of an isogenic urease-negative mutant of Helicobacter mustelaeA bifunctional urease enhances survival of pathogenic Yersinia enterocolitica and Morganella morganii at low pHMolecular aspects of bacterial pH sensing and homeostasis.Identification of low abundance microbiome in clinical samples using whole genome sequencing.Presence of active aliphatic amidases in Helicobacter species able to colonize the stomach.Gastric juice for the diagnosis of H pylori infection in patients on proton pump inhibitors.Environmental determinants of transformation efficiency in Helicobacter pylori.Gastric infection by Helicobacter pylori.Is Helicobacter pylori Infection the Primary Cause of Duodenal Ulceration or a Secondary Factor? A Review of the Evidence.Screening of 20 commonly used Iranian traditional medicinal plants against urease.Phosphorylation-dependent and Phosphorylation-independent Regulation of Helicobacter pylori Acid Acclimation by the ArsRS Two-component System.Survival of Helicobacter pylori in gastric acidic territory.Expression of the Helicobacter pylori ureI gene is required for acidic pH activation of cytoplasmic ureaseProlonged survival and cytoplasmic pH homeostasis of Helicobacter pylori at pH 1.The Helicobacter pylori UreI protein is not involved in urease activity but is essential for bacterial survival in vivo.Helicobacter pylori uses motility for initial colonization and to attain robust infectionAcidic pH changes receptor binding specificity of Helicobacter pylori: a binary adhesion model in which surface heat shock (stress) proteins mediate sulfatide recognition in gastric colonization.
P2860
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P2860
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
description
1995 nî lūn-bûn
@nan
1995 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@ast
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@en
type
label
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@ast
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@en
prefLabel
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@ast
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@en
P2093
P2860
P1476
Helicobacter pylori requires an acidic environment to survive in the presence of urea.
@en
P2093
P2860
P304
P407
P577
1995-05-01T00:00:00Z