Direct and indirect requirements of Shh/Gli signaling in early pituitary development. - Wikidata - wikimedia - TriplyDB

Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

http://www.wikidata.org/entity/Q35432502

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Fox tales: regulation of gonadotropin gene expression by forkhead transcription factors Pituitary gland development and disease: from stem cell to hormone production Ceramide is involved in alcohol-induced neural proliferation.Pituitary androgen receptor signalling regulates prolactin but not gonadotrophins in the male mouse.Prenatal ethanol exposure in mice phenocopies Cdon mutation by impeding Shh function in the etiology of optic nerve hypoplasia Disruption of SoxB1-dependent Sonic hedgehog expression in the hypothalamus causes septo-optic dysplasia.Genetic regulation of murine pituitary development.Identification of novel pathways involved in the pathogenesis of human adamantinomatous craniopharyngioma.Functional characterization of a heterozygous GLI2 missense mutation in patients with multiple pituitary hormone deficiency.Sonic Hedgehog Signaling and VACTERL Association.Deletion of OTX2 in neural ectoderm delays anterior pituitary development Inhibition of Sox2-dependent activation of Shh in the ventral diencephalon by Tbx3 is required for formation of the neurohypophysis.PROP1 triggers epithelial-mesenchymal transition-like process in pituitary stem cells SOX2 is sequentially required for progenitor proliferation and lineage specification in the developing pituitary Foxo1 Is Required for Normal Somatotrope Differentiation Notch signaling in postnatal pituitary expansion: proliferation, progenitors, and cell specification.Pathology and pathogenesis of craniopharyngiomas.Hedgehog: Multiple Paths for Multiple Roles in Shaping the Brain and Spinal Cord.Hypothalamic sonic hedgehog is required for cell specification and proliferation of LHX3/LHX4 pituitary embryonic precursors.Genetics of Combined Pituitary Hormone Deficiency: Roadmap into the Genome Era.The role of the sonic hedgehog signalling pathway in patients with midline defects and congenital hypopituitarism.Relatively high frequency of non-synonymous GLI2 variants in patients with congenital hypopituitarism without holoprosencephaly.Pathogenic mutations in GLI2 cause a specific phenotype that is distinct from holoprosencephaly.The origins of the circumventricular organs.

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P2860

Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

http://www.wikidata.org/entity/Q35432502

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

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Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

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Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

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J.YDBIO.2010.09.024

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Developmental Biology

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Direct and indirect requirements of Shh/Gli signaling in early pituitary development.

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C Brian Bai

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James F Martin

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Yiwei Wang

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P2860

Mutations in PROP1 cause familial combined pituitary hormone deficiency

Mutations in the homeobox gene HESX1/Hesx1 associated with septo-optic dysplasia in human and mouse

Multistep signaling requirements for pituitary organogenesis in vivo

Generalized lacZ expression with the ROSA26 Cre reporter strain

Reciprocal interactions of Pit1 and GATA2 mediate signaling gradient-induced determination of pituitary cell types

Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutation

Mutation of the POU-specific domain of Pit-1 and hypopituitarism without pituitary hypoplasia

Bone morphogenetic protein-4 is required for mesoderm formation and patterning in the mouse

Specific and redundant functions of Gli2 and Gli3 zinc finger genes in skeletal patterning and development

Gli1 can rescue the in vivo function of Gli2

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