Insulin promotes the biosynthesis and secretion of apolipoprotein B-48 by altering apolipoprotein B mRNA editing.
about
Phosphorylation is a regulatory mechanism in apolipoprotein B mRNA editingDevelopmental regulation of apolipoprotein B mRNA editing is an autonomous function of small intestine involving homeobox gene Cdx1Insulin enhances post-translational processing of nascent SREBP-1c by promoting its phosphorylation and association with COPII vesiclesMetabolic regulation of APOBEC-1 complementation factor trafficking in mouse models of obesity and its positive correlation with the expression of ApoB protein in hepatocytesInsulin enhances the biogenesis of nuclear sterol regulatory element-binding protein (SREBP)-1c by posttranscriptional down-regulation of Insig-2A and its dissociation from SREBP cleavage-activating protein (SCAP).SREBP-1c complex.Exploring the potential of the platelet membrane proteome as a source of peripheral biomarkers for Alzheimer's disease.Tolfenamic acid suppresses cytochrome P450 2E1 expression in mouse liverCideB protein is required for the biogenesis of very low density lipoprotein (VLDL) transport vesicleTwo distinct TATA-less promoters direct tissue-specific expression of the rat apo-B editing catalytic polypeptide 1 gene.N-3 polyunsaturated fatty acids suppress insulin-induced SREBP-1c transcription via reduced trans-activating capacity of LXRalpha.Identification of novel alternative splice variants of APOBEC-1 complementation factor with different capacities to support apolipoprotein B mRNA editing.Effects of diabetes mellitus on hepatocyte nuclear factor 1 decrease albumin gene transcription.Expression of the rat sterol regulatory element-binding protein-1c gene in response to insulin is mediated by increased transactivating capacity of specificity protein 1 (Sp1).Role of insulin in hepatic fatty acid partitioning: emerging concepts.Insulin activates the rat sterol-regulatory-element-binding protein 1c (SREBP-1c) promoter through the combinatorial actions of SREBP, LXR, Sp-1 and NF-Y cis-acting elements.Chronic exogenous hyperinsulinaemia does not modify the acute inhibitory effect of insulin on the secretion of very-low-density lipoprotein triacylglycerol and apolipoprotein B in primary cultures of rat hepatocytes.Hepatic very low density lipoprotein-ApoB overproduction is associated with attenuated hepatic insulin signaling and overexpression of protein-tyrosine phosphatase 1B in a fructose-fed hamster model of insulin resistance.Long-term growth hormone excess induces marked alterations in lipoprotein metabolism in mice.Interaction between growth hormone and insulin in the regulation of lipoprotein metabolism in the rat.Dexamethasone enhanced by insulin, but not by thyroid hormones stimulates apolipoprotein B mRNA editing in cultured rat hepatocytes depending on the developmental stage.
P2860
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P2860
Insulin promotes the biosynthesis and secretion of apolipoprotein B-48 by altering apolipoprotein B mRNA editing.
description
1994 nî lūn-bûn
@nan
1994 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@ast
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@en
type
label
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@ast
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@en
prefLabel
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@ast
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@en
P2093
P2860
P356
P1476
Insulin promotes the biosynthe ...... apolipoprotein B mRNA editing.
@en
P2093
C S Werner
F E Thorngate
H G Wilcox
M Heimberg
P2860
P304
P356
10.1073/PNAS.91.12.5392
P407
P577
1994-06-01T00:00:00Z